Variable small protein (Vsp)-dependent and Vsp-independent pathways for glycosaminoglycan recognition by relapsing fever spirochaetes. (Q40620448)

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7 November 2019

title

Variable small protein (Vsp)-dependent and Vsp-independent pathways for glycosaminoglycan recognition by relapsing fever spirochaetes (English)

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7 November 2019

7

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7 November 2019

author name string

L Magoun

1

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7 November 2019

W R Zückert

2

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7 November 2019

D Robbins

3

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7 November 2019

N Parveen

4

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7 November 2019

K R Alugupalli

5

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7 November 2019

T G Schwan

6

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7 November 2019

J M Leong

8

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7 November 2019

language of work or name

English

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publication date

1 May 2000

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7 November 2019

published in

Molecular Microbiology

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7 November 2019

volume

36

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7 November 2019

issue

4

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7 November 2019

page(s)

886-897

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Linear DNA of Borrelia species and antigenic variation

7 November 2019

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Experimental infection of the mouse brain by a relapsing fever Borrelia species: a molecular analysis

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Variability of a bacterial surface protein and disease expression in a possible mouse model of systemic Lyme borreliosis

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Integrin alpha IIb beta 3 mediates binding of the Lyme disease agent Borrelia burgdorferi to human platelets

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Genetic analysis of proteoglycan structure, function and metabolism

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The plasminogen activation system enhances brain and heart invasion in murine relapsing fever borreliosis

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Sequence changes in the pilus subunit lead to tropism variation of Neisseria gonorrhoeae to human tissue.

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Can protracted relapsing fever resemble Lyme disease?

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A single mutation affects both N-acetylglucosaminyltransferase and glucuronosyltransferase activities in a Chinese hamster ovary cell mutant defective in heparan sulfate biosynthesis

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Homology between Borrelia burgdorferi OspC and members of the family of Borrelia hermsii variable major proteins

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Identification of a candidate glycosaminoglycan-binding adhesin of the Lyme disease spirochete Borrelia burgdorferi

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Phylogenesis of relapsing fever Borrelia spp.

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Subtelomeric expression regions of Borrelia hermsii linear plasmids are highly polymorphic.

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Erythrocyte-aggregating relapsing fever spirochete Borrelia crocidurae induces formation of microemboli

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