TREM-1 expression in macrophages is regulated at transcriptional level by NF-kappaB and PU.1. (Q40106582)

17 September 2017

title

TREM-1 expression in macrophages is regulated at transcriptional level by NF-kappaB and PU.1. (English)

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17 September 2017

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Heng Zeng

1

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17 September 2017

Magdalena Ornatowska

series ordinal

2

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17 September 2017

Myung S Joo

series ordinal

3

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17 September 2017

Ruxana T Sadikot

series ordinal

4

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17 September 2017

language of work or name

English

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publication date

1 August 2007

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European Journal of Immunology

17 September 2017

published in

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17 September 2017

volume

37

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17 September 2017

issue

8

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17 September 2017

page(s)

2300-2308

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Cutting edge: inflammatory responses can be triggered by TREM-1, a novel receptor expressed on neutrophils and monocytes

17 September 2017

cites work

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Duration and intensity of NF-kappaB activity determine the severity of endotoxin-induced acute lung injury

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PU.1 regulates cathepsin S expression in professional APCs

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Lipopolysaccharide-induced up-regulation of triggering receptor expressed on myeloid cells-1 expression on macrophages is regulated by endogenous prostaglandin E2

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TREM-1 amplifies inflammation and is a crucial mediator of septic shock

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TREMs in the immune system and beyond

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TREM-1 (triggering receptor expressed on myeloid cells): a new player in acute inflammatory responses

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NF-kappaB family of transcription factors: central regulators of innate and adaptive immune functions.

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NFβA, a factor required for maximal interleukin-1β gene expression is identical to the ets family member PU.1. Evidence for structural alteration following LPS activation

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Activated Fes protein tyrosine kinase induces terminal macrophage differentiation of myeloid progenitors (U937 cells) and activation of the transcription factor PU.1.

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Transcriptional regulation of the cyclooxygenase-2 gene in macrophages by PU.1.

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Serial changes in soluble triggering receptor expressed on myeloid cells in the lung during development of ventilator-associated pneumonia

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Soluble triggering receptor expressed on myeloid cells and the diagnosis of pneumonia and severe sepsis

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Modulation of the triggering receptor expressed on the myeloid cell type 1 pathway in murine septic shock

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Soluble form of the triggering receptor expressed on myeloid cells-1 as a marker of microbial infection

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Rel/NF-kappa B/I kappa B family: intimate tales of association and dissociation

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A Toll-like receptor recognizes bacterial DNA

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Targeted immunomodulation of the NF-kappaB pathway in airway epithelium impacts host defense against Pseudomonas aeruginosa

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Requirement of transcription factor PU.1 in the development of multiple hematopoietic lineages.

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PU.1, a shared transcriptional regulator of lymphoid and myeloid cell fates

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PU.1/Interferon Regulatory Factor interactions: mechanisms of transcriptional regulation

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MIF regulates innate immune responses through modulation of Toll-like receptor 4

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7 January 2021

Identifiers

DOI

10.1002/EJI.200737270

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17 September 2017

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