Oxidation induces ClC-3-dependent anion channels in human leukaemia cells (Q40058690)

17 September 2017

title

Oxidation induces ClC-3-dependent anion channels in human leukaemia cells (English)

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17 September 2017

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Ravi S Kasinathan

1

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17 September 2017

Michael Föller

2

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17 September 2017

Camelia Lang

3

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17 September 2017

Saisudha Koka

4

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17 September 2017

Florian Lang

5

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17 September 2017

Stephan M Huber

6

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17 September 2017

language of work or name

English

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publication date

30 October 2007

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17 September 2017

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17 September 2017

volume

581

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17 September 2017

issue

28

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17 September 2017

page(s)

5407-5412

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Redox signaling in hypertension

17 September 2017

cites work

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Plasmodium induces swelling-activated ClC-2 anion channels in the host erythrocyte

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Disruption of ClC-3, a chloride channel expressed on synaptic vesicles, leads to a loss of the hippocampus

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Induction of erythroid differentiation in the human leukaemia cell line K562.

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Electrophysiological properties of the Plasmodium Falciparum-induced cation conductance of human erythrocytes

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TRPM2 is an ion channel that modulates hematopoietic cell death through activation of caspases and PARP cleavage.

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Regulation of human CLC-3 channels by multifunctional Ca2 /calmodulin-dependent protein kinase

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Regulation of intracellular Cl- concentration through volume-regulated ClC-3 chloride channels in A10 vascular smooth muscle cells

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Altered properties of volume-sensitive osmolyte and anion channels (VSOACs) and membrane protein expression in cardiac and smooth muscle myocytes from Clcn3-/- mice

21 January 2018

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Superoxide flux in endothelial cells via the chloride channel-3 mediates intracellular signaling

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Identifiers

DOI

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17 September 2017

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