The PERK/eIF2 alpha signaling pathway of Unfolded Protein Response is essential for N-(4-hydroxyphenyl)retinamide (4HPR)-induced cytotoxicity in cancer cells (Q40002218)

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English	The PERK/eIF2 alpha signaling pathway of Unfolded Protein Response is essential for N-(4-hydroxyphenyl)retinamide (4HPR)-induced cytotoxicity in cancer cells	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

16 September 2017

The PERK/eIF2 alpha signaling pathway of Unfolded Protein Response is essential for N-(4-hydroxyphenyl)retinamide (4HPR)-induced cytotoxicity in cancer cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

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author name string

Wai-Lung Lai

1

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Europe PubMed Central

PubMed publication ID

16 September 2017

Nai-Sum Wong

2

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Europe PubMed Central

PubMed publication ID

16 September 2017

language of work or name

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publication date

14 March 2008

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Europe PubMed Central

PubMed publication ID

16 September 2017

Experimental Cell Research

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Europe PubMed Central

PubMed publication ID

16 September 2017

314

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Europe PubMed Central

PubMed publication ID

16 September 2017

8

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Europe PubMed Central

PubMed publication ID

16 September 2017

1667-1682

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Europe PubMed Central

PubMed publication ID

16 September 2017

Fifteen-year results of a randomized phase III trial of fenretinide to prevent second breast cancer

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

N-(4-Hydroxyphenyl)retinamide (4-HPR) Induces Leukemia Cell Death via Generation of Reactive Oxygen Species

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Mechanism of 4-HPR-induced apoptosis in glioma cells: evidences suggesting role of mitochondrial-mediated pathway and endoplasmic reticulum stress.

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Regulation of lipocalin-2 gene by the cancer chemopreventive retinoid 4-HPR.

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https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

BBC3 mediates fenretinide-induced cell death in neuroblastoma

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Upregulation of GADD153 expression in the apoptotic signaling of N-(4-hydroxyphenyl)retinamide (4HPR).

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Fenretinide induces sustained-activation of JNK/p38 MAPK and apoptosis in a reactive oxygen species-dependent manner in neuroblastoma cells

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

N-(4-hydroxyphenyl)retinamide-induced apoptosis triggered by reactive oxygen species is mediated by activation of MAPKs in head and neck squamous carcinoma cells

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Regulated translation initiation controls stress-induced gene expression in mammalian cells

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Role of Noxa in p53-independent fenretinide-induced apoptosis of neuroectodermal tumours

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Induction of endoplasmic reticulum stress by the pro-apoptotic retinoid N-(4-hydroxyphenyl)retinamide via a reactive oxygen species-dependent mechanism in human head and neck cancer cells

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

The mammalian unfolded protein response

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Cellular response to endoplasmic reticulum stress: a matter of life or death

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Feedback inhibition of the unfolded protein response by GADD34-mediated dephosphorylation of eIF2alpha

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Identification of the cis-acting endoplasmic reticulum stress response element responsible for transcriptional induction of mammalian glucose-regulated proteins. Involvement of basic leucine zipper transcription factors

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Dimerization and release of molecular chaperone inhibition facilitate activation of eukaryotic initiation factor-2 kinase in response to endoplasmic reticulum stress

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Nod1, an Apaf-1-like activator of caspase-9 and nuclear factor-kappaB

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

ROS mediates 4HPR-induced posttranscriptional expression of the Gadd153 gene

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

ER stress signaling by regulated proteolysis of ATF6

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Regulated externalization of phosphatidylserine at the cell surface: implications for apoptosis

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Autophagy counterbalances endoplasmic reticulum expansion during the unfolded protein response

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Targeting homeostatic mechanisms of endoplasmic reticulum stress to increase susceptibility of cancer cells to fenretinide-induced apoptosis: the role of stress proteins ERdj5 and ERp57

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Diabetes mellitus and exocrine pancreatic dysfunction in perk-/- mice reveals a role for translational control in secretory cell survival

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Perk is essential for translational regulation and cell survival during the unfolded protein response

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Mechanisms of fenretinide-induced apoptosis

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

The chemopreventive agent N-(4-hydroxyphenyl)retinamide induces apoptosis through a mitochondrial pathway regulated by proteins from the Bcl-2 family

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Autophagy is activated for cell survival after endoplasmic reticulum stress

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Self-eating and self-killing: crosstalk between autophagy and apoptosis

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Novel cell death pathways induced by N-(4-hydroxyphenyl)retinamide: therapeutic implications.

1 reference

https://api.crossref.org/works/10.1016/J.YEXCR.2008.02.002

7 January 2021

Identifiers

10.1016/J.YEXCR.2008.02.002

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

16 September 2017

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