A novel promoter sequence is involved in the oxidative stress-induced expression of the adult T-cell leukemia-derived factor (ADF)/human thioredoxin (Trx) gene (Q39716872)

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scientific article published on July 1996

Language	Label	Description	Also known as
English	A novel promoter sequence is involved in the oxidative stress-induced expression of the adult T-cell leukemia-derived factor (ADF)/human thioredoxin (Trx) gene	scientific article published on July 1996

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

A novel promoter sequence is involved in the oxidative stress-induced expression of the adult T-cell leukemia-derived factor (ADF)/human thioredoxin (Trx) gene (English)

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

1 reference

based on heuristic

inferred from title

author name string

Taniguchi Y

1

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

Taniguchi-Ueda Y

2

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

Mori K

3

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

Yodoi J

4

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

language of work or name

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publication date

1 July 1996

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Europe PubMed Central

PMC publication ID

14 September 2017

Nucleic Acids Research

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Europe PubMed Central

PMC publication ID

14 September 2017

24

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

14

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

2746-2752

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

Oxidoreductive regulation of nuclear factor kappa B. Involvement of a cellular reducing catalyst thioredoxin

1 reference

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1 reference

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1 reference

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Chemical and molecular regulation of enzymes that detoxify carcinogens

1 reference

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Reactive oxygen intermediates target CC(A/T)6GG sequences to mediate activation of the early growth response 1 transcription factor gene by ionizing radiation

1 reference

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Distinct effects of thioredoxin and antioxidants on the activation of transcription factors NF-kappa B and AP-1.

1 reference

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Cloning and expression of a cDNA for human thioredoxin

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Transcriptional regulator of oxidative stress-inducible genes: direct activation by oxidation

1 reference

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Adult T-cell leukemia-derived factor/thioredoxin, produced by both human T-lymphotropic virus type I- and Epstein-Barr virus-transformed lymphocytes, acts as an autocrine growth factor and synergizes with interleukin 1 and interleukin 2

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Reactive oxygen intermediates as apparently widely used messengers in the activation of the NF-kappa B transcription factor and HIV-1

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Modulation of transcription factor NF-kappa B binding activity by oxidation-reduction in vitro

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1 reference

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ADF, a growth-promoting factor derived from adult T cell leukemia and homologous to thioredoxin: involvement in lymphocyte immortalization by HTLV-I and EBV.

1 reference

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TFD: The transcription factors database

1 reference

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Lymphocyte transformation and thiol compounds; the role of ADF/thioredoxin as an endogenous reducing agent

1 reference

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Thioredoxin regulates the DNA binding activity of NF-kappa B by reduction of a disulphide bond involving cysteine 62

1 reference

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Nuclear factor kappa B: an oxidative stress-responsive transcription factor of eukaryotic cells (a review)

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Genomic cloning of human thioredoxin-encoding gene: mapping of the transcription start point and analysis of the promoter

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Thiol-mediated redox regulation of lymphocyte proliferation. Possible involvement of adult T cell leukemia-derived factor and glutathione in transferrin receptor expression

1 reference

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Identifiers

10.1093/NAR/24.14.2746

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 September 2017

ResearchGate publication ID

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