The periodontal pathogen Porphyromonas gingivalis induces the Epstein–Barr virus lytic switch transactivator ZEBRA by histone modification (Q39426632)

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scientific article published on December 8, 2011

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English	The periodontal pathogen Porphyromonas gingivalis induces the Epstein–Barr virus lytic switch transactivator ZEBRA by histone modification	scientific article published on December 8, 2011

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scholarly article

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Europe PubMed Central

PubMed publication ID

11 September 2017

The periodontal pathogen Porphyromonas gingivalis induces the Epstein-Barr virus lytic switch transactivator ZEBRA by histone modification (English)

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Europe PubMed Central

PubMed publication ID

11 September 2017

The periodontal pathogen Porphyromonas gingivalis induces the Epstein–Barr virus lytic switch transactivator ZEBRA by histone modification (English)

1 reference

10.1016/J.BIOCHI.2011.12.001

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Porphyromonas gingivalis

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Epstein–Barr virus

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author name string

Kenichi Imai

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11 September 2017

Harumi Inoue

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11 September 2017

Muneaki Tamura

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11 September 2017

Hiroko Inoue

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11 September 2017

Osamu Takeichi

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11 September 2017

Kaoru Kusama

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11 September 2017

Ichiro Saito

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11 September 2017

Kuniyasu Ochiai

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11 September 2017

Marni E. Cueno

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC:MED AND EXT_ID:22178321

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publication date

8 December 2011

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11 September 2017

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https://api.elsevier.com/content/article/PII:S030090841100455X?httpAccept=text/xml

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10.1016/J.BIOCHI.2011.12.001

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https://api.elsevier.com/content/article/PII:S030090841100455X?httpAccept=text/plain

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10.1016/J.BIOCHI.2011.12.001

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Europe PubMed Central

PubMed publication ID

11 September 2017

94

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11 September 2017

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11 September 2017

839-846

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11 September 2017

Reactivation of Epstein-Barr virus: regulation and function of the BZLF1 gene.

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Activation of Epstein-Barr virus promoters by a growth-factor and a glucocorticoid

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Latent and lytic Epstein-Barr virus replication strategies

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Activation of expression of latent Epstein-Barr herpesvirus after gene transfer with a small cloned subfragment of heterogeneous viral DNA

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Possible involvement of EBV-mediated alpha-fodrin cleavage for organ-specific autoantigen in Sjogren's syndrome

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Histone hyperacetylation occurs on promoters of lytic cycle regulatory genes in Epstein-Barr virus-infected cell lines which are refractory to disruption of latency by histone deacetylase inhibitors

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Histone acetylation and reactivation of Epstein-Barr virus from latency.

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Epigenetic regulation of latent Epstein-Barr virus promoters

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Transcriptional repression by sumoylation of Epstein-Barr virus BZLF1 protein correlates with association of histone deacetylase

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Protein kinase C-independent activation of the Epstein-Barr virus lytic cycle

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MEF2-mediated recruitment of class II HDAC at the EBV immediate early gene BZLF1 links latency and chromatin remodeling

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Binding of the ubiquitous cellular transcription factors Sp1 and Sp3 to the ZI domains in the Epstein-Barr virus lytic switch BZLF1 gene promoter

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Cyclosporin A-sensitive induction of the Epstein-Barr virus lytic switch is mediated via a novel pathway involving a MEF2 family member

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Valproic acid enhances the efficacy of chemotherapy in EBV-positive tumors by increasing lytic viral gene expression

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Induction of the Epstein-Barr virus (EBV) cycle in latently infected cells by n-butyrate

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Arginine butyrate-induced susceptibility to ganciclovir in an Epstein-Barr-virus-associated lymphoma

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Effect of n-butyrate on cellular and viral DNA synthesis in cells latently infected with Epstein-Barr virus

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10.1016/J.BIOCHI.2011.12.001

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Periodontal diseases.

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Implantation of Bacteroides gingivalis in nonhuman primates initiates progression of periodontitis

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Treatment of periodontitis and endothelial function

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Preterm birth and periodontal disease

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Herpesviruses in human periodontal disease

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Herpesviral-bacterial synergy in the pathogenesis of human periodontitis

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Periodontopathic bacteria and herpesviruses in chronic periodontitis

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Herpesviral-bacterial interrelationships in aggressive periodontitis

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Periodontitis lesions are a source of salivary cytomegalovirus and Epstein-Barr virus

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Bacteroides (Porphyromonas) gingivalis fimbriae activate mouse peritoneal macrophages and induce gene expression and production of interleukin-1

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Reactivation of latent HIV-1 infection by the periodontopathic bacterium Porphyromonas gingivalis involves histone modification

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Epstein-Barr virus BZLF1 gene, a switch from latency to lytic infection, is expressed as an immediate-early gene after primary infection of B lymphocytes

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Glucocorticoids activate Epstein Barr virus lytic replication through the upregulation of immediate early BZLF1 gene expression

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Transforming growth factor beta 1 stimulates expression of the Epstein-Barr virus BZLF1 immediate-early gene product ZEBRA by an indirect mechanism which requires the MAPK kinase pathway

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Up regulation of the Epstein-Barr virus (EBV)-encoded membrane protein LMP in the Burkitt's lymphoma line Daudi after exposure to n-butyrate and after EBV superinfection

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Gingipains from Porphyromonas gingivalis W83 synergistically disrupt endothelial cell adhesion and can induce caspase-independent apoptosis

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Identification of a novel element involved in regulation of the lytic switch BZLF1 gene promoter of Epstein-Barr virus

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Identifiers

10.1016/J.BIOCHI.2011.12.001

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Europe PubMed Central

PubMed publication ID

11 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

11 September 2017

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