NF-κB-mediated transcriptional upregulation of TNFAIP2 by the Epstein-Barr virus oncoprotein, LMP1, promotes cell motility in nasopharyngeal carcinoma. (Q39105321)

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scientific article published on 26 August 2013

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English	NF-κB-mediated transcriptional upregulation of TNFAIP2 by the Epstein-Barr virus oncoprotein, LMP1, promotes cell motility in nasopharyngeal carcinoma.	scientific article published on 26 August 2013

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8 March 2020

NF-κB-mediated transcriptional upregulation of TNFAIP2 by the Epstein-Barr virus oncoprotein, LMP1, promotes cell motility in nasopharyngeal carcinoma (English)

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8 March 2020

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Epstein–Barr virus

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2

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8 March 2020

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C-C Chen

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8 March 2020

M Chao

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Y Liang

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N-M Tsang

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H-Y Huang

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26 August 2013

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8 March 2020

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8 March 2020

3648-3659

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8 March 2020

Trends in incidence and mortality of nasopharyngeal carcinoma over a 20-25 year period (1978/1983-2002) in Sihui and Cangwu counties in southern China

1 reference

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Epstein-Barr virus-encoded LMP1 interacts with FGD4 to activate Cdc42 and thereby promote migration of nasopharyngeal carcinoma cells

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M-Sec promotes membrane nanotube formation by interacting with Ral and the exocyst complex

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A novel role for TNFAIP2: its correlation with invasion and metastasis in nasopharyngeal carcinoma.

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Host-tumor interactions in nasopharyngeal carcinomas

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Epstein-Barr virus infection alters cellular signal cascades in human nasopharyngeal epithelial cells

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Epstein-Barr virus latent membrane protein 1 induces the chemotherapeutic target, thymidine phosphorylase, via NF-kappaB and p38 MAPK pathways

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Membrane nanotubes: dynamic long-distance connections between animal cells

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Functional connectivity between immune cells mediated by tunneling nanotubules

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Membrane nanotubes physically connect T cells over long distances presenting a novel route for HIV-1 transmission

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