MicroRNA-106b-5p boosts glioma tumorigensis by targeting multiple tumor suppressor genes (Q39069313)

8 September 2017

title

MicroRNA-106b-5p boosts glioma tumorigensis by targeting multiple tumor suppressor genes (English)

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8 September 2017

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F Liu

1

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8 September 2017

J Gong

2

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8 September 2017

W Huang

3

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8 September 2017

Z Wang

4

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8 September 2017

M Wang

5

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8 September 2017

J Yang

6

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8 September 2017

C Wu

7

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8 September 2017

Z Wu

8

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8 September 2017

B Han

9

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8 September 2017

language of work or name

English

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publication date

28 October 2013

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8 September 2017

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8 September 2017

volume

33

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8 September 2017

issue

40

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8 September 2017

page(s)

4813-4822

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The 2007 WHO classification of tumours of the central nervous system

8 September 2017

cites work

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MiR-106b promotes cell proliferation via targeting RB in laryngeal carcinoma

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Down‐regulation of microRNA 106b is involved in p21‐mediated cell cycle arrest in response to radiation in prostate cancer cells

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Weak seed-pairing stability and high target-site abundance decrease the proficiency of lsy-6 and other microRNAs

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Potent Anti-Tumor Effect Generated by a Novel Human Papillomavirus (HPV) Antagonist Peptide Reactivating the pRb/E2F Pathway

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Genomic profiling of microRNA and messenger RNA reveals deregulated microRNA expression in prostate cancer

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The microbe-derived short chain fatty acid butyrate targets miRNA-dependent p21 gene expression in human colon cancer

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MicroRNAs in the miR-106b family regulate p21/CDKN1A and promote cell cycle progression

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The miR-106b-25 polycistron, activated by genomic amplification, functions as an oncogene by suppressing p21 and Bim

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miR-106b aberrantly expressed in a double transgenic mouse model for Alzheimer's disease targets TGF-β type II receptor

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MicroRNA-106b-25 cluster expression is associated with early disease recurrence and targets caspase-7 and focal adhesion in human prostate cancer.

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MiR-20a and miR-106b negatively regulate autophagy induced by leucine deprivation via suppression of ULK1 expression in C2C12 myoblasts

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MicroRNAs MiR-17, MiR-20a, and MiR-106b act in concert to modulate E2F activity on cell cycle arrest during neuronal lineage differentiation of USSC.

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Caspase 8 is absent or low in many ex vivo gliomas.

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Isolation and culture of human astrocytes

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A system for stable expression of short interfering RNAs in mammalian cells

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MicroRNA sponges: competitive inhibitors of small RNAs in mammalian cells

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Upregulation of miR-20a and miR-106b is involved in the acquisition of malignancy of pediatric brainstem gliomas

21 January 2018

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31 March 2020

Identifiers

DOI

10.1038/ONC.2013.428

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8 September 2017

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