A Nonadjuvanted IgG2a Monoclonal Antibody against Nucleosomes Elicits Potent T Cell-Dependent, Idiotype-Specific IgG1 Responses and Glomerular IgG1/IgG2a Deposits in Normal Mice (Q38737661)

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scientific article published on 07 June 2017

Language	Label	Description	Also known as
English	A Nonadjuvanted IgG2a Monoclonal Antibody against Nucleosomes Elicits Potent T Cell-Dependent, Idiotype-Specific IgG1 Responses and Glomerular IgG1/IgG2a Deposits in Normal Mice	scientific article published on 07 June 2017

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

6 September 2017

A Nonadjuvanted IgG2a Monoclonal Antibody against Nucleosomes Elicits Potent T Cell-Dependent, Idiotype-Specific IgG1 Responses and Glomerular IgG1/IgG2a Deposits in Normal Mice (English)

1 reference

Europe PubMed Central

PubMed publication ID

6 September 2017

author name string

Kristian Hannestad

1

1 reference

Europe PubMed Central

PubMed publication ID

6 September 2017

Helge Scott

2

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Europe PubMed Central

PubMed publication ID

6 September 2017

language of work or name

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publication date

7 June 2017

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Europe PubMed Central

PubMed publication ID

6 September 2017

Journal of Immunology

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Europe PubMed Central

PubMed publication ID

6 September 2017

199

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Europe PubMed Central

PubMed publication ID

6 September 2017

2

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Europe PubMed Central

PubMed publication ID

6 September 2017

489-500

1 reference

Europe PubMed Central

PubMed publication ID

6 September 2017

Autoimmune-like antibodies to the ligand-binding sites of myeloma proteins

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Myeloma proteins as tumor-specific transplantation antigens

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Cell-Bound Myeloma Proteins on the Surface of Myeloma Cells: Potential Targets for the Immune System

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Antibodies to idiotypes of isologous immunoglobulins

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H--2-linked genes control immune response to V-domains of myeloma protein 315.

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H-2-linked Ir genes have a striking influence on the immunogenicity of idiotopes of myeloma protein 315 for T helper cells

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Immunological paralysis induced by an idiotypic antigen.

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Anti-idiotypic immune responses against adjuvant-free isologous IgM monoclonal antibodies and their augmentation by complex formation between IgM and albumin in bovine serum

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A syngeneic idiotype is immunogenic when borne by IgM but tolerogenic when joined to IgG.

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Chromatin-IgG complexes activate B cells by dual engagement of IgM and Toll-like receptors

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A Toll-like receptor recognizes bacterial DNA

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Antinucleosome autoantibodies bind directly to cell lines in vitro and via the FcgammaRIIB receptor to B lymphocytes in vivo: a role for immune complexes in interactions between antinucleosome IgG2a and B cells of BXSB lupus mice

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Growth of cells in a new defined protein-free medium

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Use of selectively trypsinized nucleosome core particles to analyze the role of the histone "tails" in the stabilization of the nucleosome

1 reference

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Helper T cell recognition of the variable domains of a mouse myeloma protein (315). Effect of the major histocompatibility complex and domain conformation

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Cellular sites of immunologic unresponsiveness

1 reference

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Production of BALB/c Anti-Idiotypic Antibodies Against the BALB/c Myeloma Protein 315 Does Not Require an Intact Ligand-Binding Site

1 reference

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Regulation of antibody responses via antibodies, complement, and Fc receptors

1 reference

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Responsiveness of B cells is regulated by the hinge region of IgD.

1 reference

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Monovalent ligation of the B cell receptor induces receptor activation but fails to promote antigen presentation.

1 reference

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Fcgamma receptor-mediated induction of dendritic cell maturation and major histocompatibility complex class I-restricted antigen presentation after immune complex internalization

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Somatic hypermutation as a generator of antinuclear antibodies in a murine model of systemic autoimmunity

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Anti-DNA antibodies from autoimmune mice arise by clonal expansion and somatic mutation

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Both IgM and IgG anti-DNA antibodies are the products of clonally selective B cell stimulation in (NZB x NZW)F1 mice

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Adjuvant-enhanced antibody responses in the absence of toll-like receptor signaling

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B cell intrinsic TLR signals amplify but are not required for humoral immunity

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Innate immune recognition

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Nucleosomes are exposed at the cell surface in apoptosis.

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Natural antibody to apoptotic cell membranes inhibits the proinflammatory properties of lupus autoantibody immune complexes.

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Soluble antigen can cause enhanced apoptosis of germinal-centre B cells.

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Antigen-induced B-cell death and elimination during germinal-centre immune responses

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Identifiers

10.4049/JIMMUNOL.1600718

1 reference

Europe PubMed Central

PubMed publication ID

6 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 September 2017

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