Upregulation of thioredoxin-1 in activated human NK cells confers increased tolerance to oxidative stress. (Q38715422)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:28224212 AND SRC:MED&resulttype=core&format=json

3 April 2020

title

Upregulation of thioredoxin-1 in activated human NK cells confers increased tolerance to oxidative stress (English)

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3 April 2020

7

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3 April 2020

Jimmy So

8

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3 April 2020

author name string

Kousaku Mimura

1

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3 April 2020

Ley-Fang Kua

2

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3 April 2020

Noriko Shimasaki

3

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3 April 2020

Kensuke Shiraishi

4

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3 April 2020

Shotaro Nakajima

5

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3 April 2020

Lim Kee Siang

6

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3 April 2020

Wei-Peng Yong

9

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3 April 2020

Koji Kono

10

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3 April 2020

publication date

21 February 2017

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Cancer Immunology, Immunotherapy

3 April 2020

published in

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3 April 2020

volume

66

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3 April 2020

issue

5

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3 April 2020

page(s)

605-613

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Nivolumab in previously untreated melanoma without BRAF mutation

3 April 2020

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T cells expanded in presence of IL-15 exhibit increased antioxidant capacity and innate effector molecules

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Interleukin-2 Receptor Signaling: At the Interface between Tolerance and Immunity

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Increased thioredoxin-1 production in human naturally occurring regulatory T cells confers enhanced tolerance to oxidative stress.

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Activation of the PI3K/mTOR pathway by BCR-ABL contributes to increased production of reactive oxygen species

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Adenanthin, a new inhibitor of thiol-dependent antioxidant enzymes, impairs the effector functions of human natural killer cells

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Overexpression of human thioredoxin in transgenic mice controls oxidative stress and life span.

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