Molecular basis of funny current (If) in normal and failing human heart. (Q38551800)

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9 January 2020

title

Molecular basis of funny current (If) in normal and failing human heart (English)

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9 January 2020

1

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9 January 2020

Elisabetta Cerbai

5

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9 January 2020

Stanley Nattel

7

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9 January 2020

author name string

Giuseppe Lonardo

2

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9 January 2020

Stephen Zicha

3

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9 January 2020

Andras Varro

4

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Alessandro Mugelli

6

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publication date

11 May 2008

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Journal of Molecular and Cellular Cardiology

9 January 2020

published in

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volume

45

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issue

2

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9 January 2020

page(s)

289-299

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The epidemiology of heart failure

9 January 2020

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The Properties of the Pacemaker Current IFin Human Ventricular Myocytes are Modulated by Cardiac Disease

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Hyperpolarization-Activated Inward Current in Ventricular Myocytes From Normal and Failing Human Hearts

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Abnormal diastolic currents in ventricular myocytes from spontaneous hypertensive heart failure rats

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Occurrence and properties of the hyperpolarization-activated current If in ventricular myocytes from normotensive and hypertensive rats during aging

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Characterization of the hyperpolarization-activated current, I(f), in ventricular myocytes from human failing heart

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Hyperpolarization-activated cyclic nucleotide-gated channel 1 is a molecular determinant of the cardiac pacemaker current I(f).

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Molecular basis of species-specific expression of repolarizing K currents in the heart.

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Role of subunit heteromerization and N-linked glycosylation in the formation of functional hyperpolarization-activated cyclic nucleotide-gated channels

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Characterization of the hyperpolarization-activated current, I(f), in ventricular myocytes isolated from hypertensive rats

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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Measurement of funny current (I(f)) channel mRNA in human atrial tissue: correlation with left atrial filling pressure and atrial fibrillation

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KCNE2 protein is expressed in ventricles of different species, and changes in its expression contribute to electrical remodeling in diseased hearts

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Molecular characterization of a slowly gating human hyperpolarization-activated channel predominantly expressed in thalamus, heart, and testis

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The HCN gene family: molecular basis of the hyperpolarization-activated pacemaker channels

7 January 2021

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Properties of hyperpolarization-activated pacemaker current defined by coassembly of HCN1 and HCN2 subunits and basal modulation by cyclic nucleotide

7 January 2021

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Heteromeric HCN1-HCN4 channels: a comparison with native pacemaker channels from the rabbit sinoatrial node

7 January 2021

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MiRP1 modulates HCN2 channel expression and gating in cardiac myocytes

7 January 2021

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Localization of pacemaker channels in lipid rafts regulates channel kinetics

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7 January 2021

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7 January 2021

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Regional distribution of hyperpolarization‐activated current (If) and hyperpolarization‐activated cyclic nucleotide‐gated channel mRNA expression in ventricular cells from control and hypertrophied rat hearts

7 January 2021

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733 A molecular basis for HCN channel over-expression

7 January 2021

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Single-channel properties support a potential contribution of hyperpolarization-activated cyclic nucleotide-gated channels and If to cardiac arrhythmias

7 January 2021

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Arrhythmogenic ion-channel remodeling in the heart: heart failure, myocardial infarction, and atrial fibrillation.

7 January 2021

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7 January 2021

Identifiers

DOI

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9 January 2020

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