A disintegrin and metalloprotease with thrombospondin type I motif 7: a new protease for connective tissue growth factor in hepatic progenitor/oval cell niche. (Q35669047)

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scientific article published on 2 April 2015

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English	A disintegrin and metalloprotease with thrombospondin type I motif 7: a new protease for connective tissue growth factor in hepatic progenitor/oval cell niche.	scientific article published on 2 April 2015

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4 March 2020

A disintegrin and metalloprotease with thrombospondin type I motif 7: a new protease for connective tissue growth factor in hepatic progenitor/oval cell niche (English)

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connective tissue

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Liya Pi

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Seh-Hoon Oh

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Yianni Protopapadakis

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Altin Gjymishka

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Alicia Brown

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Paulette Robinson

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Edward W Scott

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Gregory S Schultz

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Bryon E Petersen

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The American Journal of Pathology

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1552-1563

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Bipotential adult liver progenitors are derived from chronically injured mature hepatocytes

1 reference

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Connective tissue growth factor and integrin αvβ6: a new pair of regulators critical for ductular reaction and biliary fibrosis in mice

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Evidence against a stem cell origin of new hepatocytes in a common mouse model of chronic liver injury

1 reference

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12 July 2018

Adult hepatocytes are generated by self-duplication rather than stem cell differentiation.

1 reference

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Clonal tracing of Sox9 liver progenitors in mouse oval cell injury

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Links between hepatic fibrosis, ductular reaction, and progenitor cell expansion

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Biochemistry and physiological functions of ADAMTS7 metalloprotease

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Macrophage-derived Wnt opposes Notch signaling to specify hepatic progenitor cell fate in chronic liver disease.

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Loss of matrix metalloproteinase-2 amplifies murine toxin-induced liver fibrosis by upregulating collagen I expression

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The role of stem cells in liver repair and fibrosis

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12 July 2018

Characterisation of the liver progenitor cell niche in liver diseases: potential involvement of Wnt and Notch signalling

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12 July 2018

A disintegrin-like and metalloprotease (reprolysin-type) with thrombospondin type 1 motif (ADAMTS) superfamily: functions and mechanisms

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12 July 2018

ADAMTS-7, a direct target of PTHrP, adversely regulates endochondral bone growth by associating with and inactivating GEP growth factor

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Connective tissue growth factor: a fibrogenic master switch in fibrotic liver diseases

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RNA interfering connective tissue growth factor prevents rat hepatic stellate cell activation and extracellular matrix production

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Inhibition of ADAMTS-7 and ADAMTS-12 degradation of cartilage oligomeric matrix protein by alpha-2-macroglobulin

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Connective tissue growth factor with a novel fibronectin binding site promotes cell adhesion and migration during rat oval cell activation

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The ADAMTS metalloproteinases

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Remodelling of extracellular matrix is a requirement for the hepatic progenitor cell response

1 reference

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Protease profiling of liver fibrosis reveals the ADAM metallopeptidase with thrombospondin type 1 motif, 1 as a central activator of transforming growth factor beta

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Mechanisms and therapy of hepatic fibrosis: report of the AASLD Single Topic Basic Research Conference

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Cholestatic liver fibrosis and toxin-induced fibrosis are exacerbated in matrix metalloproteinase-2 deficient mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/25843683

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based on heuristic

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10.1016/J.AJPATH.2015.02.008

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4 March 2020

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4 March 2020

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