Time-course of nigrostriatal degeneration in a progressive MPTP-lesioned macaque model of Parkinson's disease (Q35622290)

9 August 2017

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title

Time-course of nigrostriatal degeneration in a progressive MPTP-lesioned macaque model of Parkinson's disease (English)

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9 August 2017

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Denis Guilloteau

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Wassilios G. Meissner

object named as

Wassilios Meissner

1

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Sylvie Chalon

object named as

Sylvie Chalon

4

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Erwan Bezard

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Erwan Bezard

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author name string

Caroline Prunier

2

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9 August 2017

Christian E Gross

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9 August 2017

publication date

1 December 2003

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9 August 2017

published in

Molecular Neurobiology

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9 August 2017

volume

28

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9 August 2017

issue

3

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9 August 2017

page(s)

209-218

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Longitudinal fluorodopa positron emission tomographic studies of the evolution of idiopathic parkinsonism

9 August 2017

cites work

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Parkinsonism: onset, progression, and mortality

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Time course of nigrostriatal degeneration in parkinson's disease. A detailed study of influential factors in human brain amine analysis

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Rate of cell death in parkinsonism indicates active neuropathological process

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Axonal self-destruction and neurodegeneration

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Melanized dopaminergic neurons are differentially susceptible to degeneration in Parkinson's disease

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Presymptomatic diagnosis of experimental Parkinsonism with 123I-PE2I SPECT.

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Pattern of levodopa-induced striatal changes is different in normal and MPTP-lesioned mice.

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Differential vulnerability of primate caudate-putamen and striosome-matrix dopamine systems to the neurotoxic effects of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine

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Chronic systemic pesticide exposure reproduces features of Parkinson's disease

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Uneven pattern of dopamine loss in the striatum of patients with idiopathic Parkinson's disease. Pathophysiologic and clinical implications

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A mathematical model of pathogenesis in idiopathic parkinsonism

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1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced parkinsonian syndrome in Macaca fascicularis: which midbrain dopaminergic neurons are lost?

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Striatal dopaminergic correlates of stable parkinsonism and degree of recovery in old-world primates one year after MPTP treatment

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Structures outside the basal ganglia may compensate for dopamine loss in the presymptomatic stages of Parkinson's disease.

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MPTP-induced parkinsonism in primates: pattern of striatal dopamine loss following acute and chronic administration.

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Primate model of parkinsonism: selective lesion of nigrostriatal neurons by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine produces an extrapyramidal syndrome in rhesus monkeys

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Chronic exposure to low doses of MPTP. I. Cognitive deficits in motor asymptomatic monkeys

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A chronic MPTP model reproducing the slow evolution of Parkinson's disease: evolution of motor symptoms in the monkey

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Compensatory mechanisms in experimental and human parkinsonism: towards a dynamic approach

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Glial cells and inflammation in Parkinson's disease: a role in neurodegeneration?

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Mechanism and consequences of nerve cell death in Parkinson's disease.

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The neurotoxin MPTP causes degeneration of specific nucleus A8, A9 and A10 dopaminergic neurons in the mouse

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The evolution of nigrostriatal neurochemical changes in the MPTP-treated squirrel monkey

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Dopamine transporter is required for in vivo MPTP neurotoxicity: evidence from mice lacking the transporter.

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Stable parkinsonian syndrome and uneven loss of striatal dopamine fibres following chronic MPTP administration in baboons

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Relationship between the appearance of symptoms and the level of nigrostriatal degeneration in a progressive 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-lesioned macaque model of Parkinson's disease.

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Clinical observations on the rate of progression of idiopathic parkinsonism

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Chronic systemic complex I inhibition induces a hypokinetic multisystem degeneration in rats

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Striatal preproenkephalin gene expression is upregulated in acute but not chronic parkinsonian monkeys: implications for the contribution of the indirect striatopallidal circuit to parkinsonian symptomatology

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Evidence of active nerve cell degeneration in the substantia nigra of humans years after 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine exposure

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Chronic MPTP treatment reproduces in baboons the differential vulnerability of mesencephalic dopaminergic neurons observed in Parkinson's disease

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Ageing and Parkinson's disease: substantia nigra regional selectivity

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Differing patterns of striatal 18F-dopa uptake in Parkinson's disease, multiple system atrophy, and progressive supranuclear palsy

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Brain dopamine and the syndromes of Parkinson and Huntington. Clinical, morphological and neurochemical correlations

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The substantia nigra of the human brain. II. Patterns of loss of dopamine-containing neurons in Parkinson's disease

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An [18F]dopa-PET and clinical study of the rate of progression in Parkinson's disease

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Upregulation of striatal preproenkephalin gene expression occurs before the appearance of parkinsonian signs in 1-methyl-4-phenyl- 1,2,3,6-tetrahydropyridine monkeys

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Chronic Parkinsonism in humans due to a product of meperidine-analog synthesis

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12 December 2020

Pattern of dopaminergic loss in the striatum of humans with MPTP induced parkinsonism

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12 December 2020

Does a long preclinical period occur in Parkinson's disease?

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12 December 2020

Identifiers

DOI

10.1385/MN:28:3:209

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Dimensions Publication ID

9 August 2017

1015551492

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