Post-treatment with an ultra-low dose of NADPH oxidase inhibitor diphenyleneiodonium attenuates disease progression in multiple Parkinson's disease models (Q35529695)

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scientific article published on 25 February 2015

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English	Post-treatment with an ultra-low dose of NADPH oxidase inhibitor diphenyleneiodonium attenuates disease progression in multiple Parkinson's disease models	scientific article published on 25 February 2015

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1 March 2020

Post-treatment with an ultra-low dose of NADPH oxidase inhibitor diphenyleneiodonium attenuates disease progression in multiple Parkinson's disease models (English)

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1 March 2020

Parkinson's disease

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Jau-Shyong Hong

10

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1 March 2020

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Qingshan Wang

1

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1 March 2020

Li Qian

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1 March 2020

Shih-Heng Chen

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1 March 2020

Chun-Hsien Chu

4

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Belinda Wilson

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1 March 2020

Esteban Oyarzabal

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1 March 2020

Syed Ali

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1 March 2020

Bonnie Robinson

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1 March 2020

Deepa Rao

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25 February 2015

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1 March 2020

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1 March 2020

138

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1 March 2020

Pt 5

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1 March 2020

1247-1262

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1 March 2020

Substance P exacerbates dopaminergic neurodegeneration through neurokinin-1 receptor-independent activation of microglial NADPH oxidase

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Subsite awareness in neuropathology evaluation of National Toxicology Program (NTP) studies: a review of select neuroanatomical structures with their functional significance in rodents

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NADPH oxidase and aging drive microglial activation, oxidative stress, and dopaminergic neurodegeneration following systemic LPS administration

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NADPH oxidases: novel therapeutic targets for neurodegenerative diseases

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HMGB1 acts on microglia Mac1 to mediate chronic neuroinflammation that drives progressive neurodegeneration.

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Neuroinflammation and α-synuclein dysfunction potentiate each other, driving chronic progression of neurodegeneration in a mouse model of Parkinson's disease.

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Neurotoxic in vivo models of Parkinson's disease recent advances.

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Microglia in neurodegenerative disease

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Macrophage antigen complex-1 mediates reactive microgliosis and progressive dopaminergic neurodegeneration in the MPTP model of Parkinson's disease

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Classical inhibitors of NOX NAD(P)H oxidases are not specific.

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Why neurodegenerative diseases are progressive: uncontrolled inflammation drives disease progression.

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Endotoxin induces a delayed loss of TH-IR neurons in substantia nigra and motor behavioral deficits

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Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration

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Microglia-mediated neurotoxicity: uncovering the molecular mechanisms

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27 July 2018

Mechanisms of Parkinson's disease linked to pathological alpha-synuclein: new targets for drug discovery

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NSAIDs and Alzheimer disease: epidemiological, animal model and clinical studies

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Microglia and inflammation-mediated neurodegeneration: multiple triggers with a common mechanism

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Neuroprotective effect of dextromethorphan in the MPTP Parkinson's disease model: role of NADPH oxidase

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Microglial response to brain injury: a brief synopsis

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Do Compensatory Processes Underlie the Preclinical Phase of Neurodegenerative Disease? Insights from an Animal Model of Parkinsonism

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Suppression of inflammatory arthritis by simultaneous inhibition of nitric oxide synthase and NADPH oxidase.

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Compensations after lesions of central dopaminergic neurons: some clinical and basic implications

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Subpicomolar diphenyleneiodonium inhibits microglial NADPH oxidase with high specificity and shows great potential as a therapeutic agent for neurodegenerative diseases

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Advances in the pharmacological treatment of Parkinson's disease: targeting neurotransmitter systems

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The immunology of neurodegeneration

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Neurodegeneration and Inflammation in Parkinson's disease

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Current management of Parkinson's disease

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26 September 2018

Neuroprotection by caffeine: time course and role of its metabolites in the MPTP model of Parkinson's disease

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26 September 2018

Reactive microgliosis: extracellular micro-calpain and microglia-mediated dopaminergic neurotoxicity.

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Some aspects of the pharmacology of diphenyleneiodonium, a bivalent iodine compound

1 reference

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12 December 2020

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Role of reactive O2 in phagocyte-induced hypermetabolism and pulmonary injury

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https://pubmed.ncbi.nlm.nih.gov/25716193

12 December 2020

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10.1093/BRAIN/AWV034

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1 March 2020

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1 March 2020

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1 March 2020

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