IL-17 and IL-23 in lupus nephritis - association to histopathology and response to treatment (Q35078183)

6 August 2017

title

IL-17 and IL-23 in lupus nephritis - association to histopathology and response to treatment (English)

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6 August 2017

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5

Vivianne Malmström

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6 August 2017

2

Petra Amoudruz

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6 August 2017

4

Johan Rönnelid

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6 August 2017

3

Yvonne Sundström

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6 August 2017

Agneta Zickert

1

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6 August 2017

Iva Gunnarsson

series ordinal

6

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6 August 2017

publication date

12 February 2015

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6 August 2017

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6 August 2017

volume

16

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6 August 2017

page(s)

7

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Creative Commons Attribution 4.0 International

6 August 2017

copyright license

start time

12 February 2015

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

0 references

cites work

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Interleukin 17 acts in synergy with B cell-activating factor to influence B cell biology and the pathophysiology of systemic lupus erythematosus.

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Expanded double negative T cells in patients with systemic lupus erythematosus produce IL-17 and infiltrate the kidneys

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Current treatment of lupus nephritis

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Hyperproduction of IL-23 and IL-17 in patients with systemic lupus erythematosus: implications for Th17-mediated inflammation in auto-immunity.

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IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

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Elevation of proinflammatory cytokine (IL-18, IL-17, IL-12) and Th2 cytokine (IL-4) concentrations in patients with systemic lupus erythematosus

21 January 2018

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BILAG 2004. Development and initial validation of an updated version of the British Isles Lupus Assessment Group's disease activity index for patients with systemic lupus erythematosus

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Identifiers

DOI

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Dimensions Publication ID

6 August 2017

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6 August 2017

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