Induction of laminitis by prolonged hyperinsulinaemia in clinically normal ponies (Q34667395)

3 August 2017

title

Induction of laminitis by prolonged hyperinsulinaemia in clinically normal ponies (English)

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3 August 2017

Katie E Asplin

1

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3 August 2017

Martin N Sillence

series ordinal

2

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3 August 2017

Christopher C Pollitt

series ordinal

3

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3 August 2017

Catherine M McGowan

series ordinal

4

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3 August 2017

publication date

24 August 2007

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3 August 2017

published in

The Veterinary Journal

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3 August 2017

volume

174

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3 August 2017

issue

3

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3 August 2017

page(s)

530-535

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Hyperinsulinemia produces both sympathetic neural activation and vasodilation in normal humans

3 August 2017

cites work

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Insulin-mediated skeletal muscle vasodilation contributes to both insulin sensitivity and responsiveness in lean humans

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Venous oxygenation in the diabetic neuropathic foot: evidence of arteriovenous shunting?

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Improved insulin sensitivity in hyperinsulinaemic ponies through physical conditioning and controlled feed intake

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Equine laminitis: glucose deprivation and MMP activation induce dermo-epidermal separation in vitro.

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Black walnut (Juglans nigra) toxicosis: a model for equine laminitis

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Activation of extracellular matrix metalloproteinases in equine laminitis

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Expression of endothelin in equine laminitis

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Measurement of basal serum insulin concentration in the diagnosis of Cushing's disease in ponies

7 January 2021

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7 January 2021

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7 January 2021

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Insulin-mediated skeletal muscle vasodilation is nitric oxide dependent. A novel action of insulin to increase nitric oxide release

7 January 2021

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Evaluation of genetic and metabolic predispositions and nutritional risk factors for pasture-associated laminitis in ponies

7 January 2021

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Insulin resistance in equids: possible role in laminitis

7 January 2021

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10.1016/J.TVJL.2007.07.003

7 January 2021

Identifiers

DOI

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3 August 2017

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