Tumor-derived G-CSF facilitates neoplastic growth through a granulocytic myeloid-derived suppressor cell-dependent mechanism (Q34081723)

30 July 2017

title

Tumor-derived G-CSF facilitates neoplastic growth through a granulocytic myeloid-derived suppressor cell-dependent mechanism (English)

1 reference

30 July 2017

Jeremy D Waight

1

1 reference

30 July 2017

Qiang Hu

2

1 reference

30 July 2017

Austin Miller

3

1 reference

30 July 2017

Song Liu

4

1 reference

30 July 2017

Scott I Abrams

5

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30 July 2017

language of work or name

English

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publication date

16 November 2011

1 reference

30 July 2017

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30 July 2017

volume

6

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30 July 2017

issue

11

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30 July 2017

page(s)

e27690

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Creative Commons Attribution 4.0 International

30 July 2017

copyright license

start time

16 November 2011

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

0 references

describes a project that uses

limma

object named as

Limma

2 references

22 June 2022

https://europepmc.org/article/PMC/3218014

inferred from PubMed Central ID database lookup

CZ Software Mentions: A large dataset of software mentions in the biomedical literature

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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5 July 2018

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Myeloid-derived suppressor cells in mammary tumor progression in FVB Neu transgenic mice.

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28 October 2018

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Activated granulocytes and granulocyte-derived hydrogen peroxide are the underlying mechanism of suppression of t-cell function in advanced cancer patients.

28 October 2018

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12 December 2020

Altered immune function during long-term host-tumor interactions can be modulated to retard autochthonous neoplastic growth

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12 December 2020

Expansion of myeloid immune suppressor Gr CD11b cells in tumor-bearing host directly promotes tumor angiogenesis

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12 December 2020

Increased Level of both CD4 FOXP3 Regulatory T Cells and CD14 HLA-DR−/low Myeloid-Derived Suppressor Cells and Decreased Level of Dendritic Cells in Patients with Multiple Myeloma

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12 December 2020

10.1371/JOURNAL.PONE.0027690

Identifiers

DOI

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30 July 2017

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30 July 2017

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