Apoptosis as a common bacterial virulence strategy (Q34067488)

30 July 2017

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title

Apoptosis as a common bacterial virulence strategy (English)

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30 July 2017

Monack D

1

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30 July 2017

Falkow S

series ordinal

2

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30 July 2017

publication date

1 March 2000

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International Journal of Medical Microbiology

30 July 2017

published in

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30 July 2017

volume

290

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30 July 2017

issue

1

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30 July 2017

page(s)

7-13

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Arthritis Associated with Yersinia Enterocolitica Infection

30 July 2017

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Apoptosis induced by bacterial pathogens

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Characterization of the operon encoding the YpkA Ser/Thr protein kinase and the YopJ protein of Yersinia pseudotuberculosis

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Apoptosis of mouse dendritic cells is triggered by listeriolysin, the major virulence determinant of Listeria monocytogenes

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Genes encoding putative effector proteins of the type III secretion system of Salmonella pathogenicity island 2 are required for bacterial virulence and proliferation in macrophages

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Functional conservation of the Salmonella and Shigella effectors of entry into epithelial cells

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The Salmonella invasin SipB induces macrophage apoptosis by binding to caspase-1

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Yersiniasignals macrophages to undergo apoptosis and YopJ is necessary for this cell death

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Yersinia-induced apoptosis in vivo aids in the establishment of a systemic infection of mice

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The cytotoxic protein YopE of Yersinia obstructs the primary host defence

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The yopJ locus is required for Yersinia-mediated inhibition of NF-kappaB activation and cytokine expression: YopJ contains a eukaryotic SH2-like domain that is essential for its repressive activity

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7 January 2021

Interleukin 1 is released by murine macrophages during apoptosis induced by Shigella flexneri

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Shigella flexneri induces apoptosis in infected macrophages

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7 January 2021

Perspectives series: host/pathogen interactions. Apoptosis in bacterial pathogenesis

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7 January 2021

Apoptosis as a proinflammatory event: what can we learn from bacteria-induced cell death?

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7 January 2021

10.1016/S1438-4221(00)80096-X

Identifiers

DOI

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