Changes to euchromatin on LAT and ICP4 following reactivation are more prevalent in an efficiently reactivating strain of HSV-1. (Q33747905)

29 July 2017

title

Changes to euchromatin on LAT and ICP4 following reactivation are more prevalent in an efficiently reactivating strain of HSV-1. (English)

1 reference

29 July 2017

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Clinton C Creech

1

1 reference

29 July 2017

Donna M Neumann

series ordinal

2

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29 July 2017

language of work or name

English

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publication date

4 November 2010

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29 July 2017

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29 July 2017

volume

5

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29 July 2017

issue

11

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29 July 2017

page(s)

e15416

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Creative Commons Attribution 4.0 International

29 July 2017

copyright license

start time

4 November 2010

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April 2022 Public Data File from Crossref

copyright status

copyrighted

0 references

cites work

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21 June 2018

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21 June 2018

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During lytic infection herpes simplex virus type 1 is associated with histones bearing modifications that correlate with active transcription.

21 June 2018

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VP16-dependent association of chromatin-modifying coactivators and underrepresentation of histones at immediate-early gene promoters during herpes simplex virus infection

21 June 2018

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21 June 2018

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21 June 2018

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Active genes are tri-methylated at K4 of histone H3

21 June 2018

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Herpes simplex virus type 1 latency-associated transcript gene promotes neuronal survival

21 June 2018

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ICP0, ICP4, or VP16 expressed from adenovirus vectors induces reactivation of latent herpes simplex virus type 1 in primary cultures of latently infected trigeminal ganglion cells

21 June 2018

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ICP0 is required for efficient reactivation of herpes simplex virus type 1 from neuronal latency

21 June 2018

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21 June 2018

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21 June 2018

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21 June 2018

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21 June 2018

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The herpes simplex virus type 1 immediate-early protein ICP0 is necessary for the efficient establishment of latent infection

21 June 2018

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21 June 2018

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21 June 2018

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The herpes simplex virus type 1 latency-associated transcript gene regulates the establishment of latency

21 June 2018

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Role of cis-acting sequences of the ICPO promoter of herpes simplex virus type 1 in viral pathogenesis, latency and reactivation

21 June 2018

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A 348-base-pair region in the latency-associated transcript facilitates herpes simplex virus type 1 reactivation.

21 June 2018

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2973973

The herpes simplex virus type 1 regulatory protein ICP0 enhances virus replication during acute infection and reactivation from latency.

21 June 2018

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Strain specificity of spontaneous and adrenergically induced HSV-1 ocular reactivation in latently infected rabbits

21 June 2018

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21 June 2018

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During latency, herpes simplex virus type 1 DNA is associated with nucleosomes in a chromatin structure

21 June 2018

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Immediate-early regulatory gene mutants define different stages in the establishment and reactivation of herpes simplex virus latency

21 June 2018

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Herpes simplex virus type 1 ICP0 regulates expression of immediate-early, early, and late genes in productively infected cells

21 June 2018

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Acetylation of histone H3 and adrenergic-regulated gene transcription in rat pinealocytes

21 June 2018

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Crossref

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Reactivation of expression from quiescent herpes simplex virus type 1 genomes in the absence of immediate-early protein ICP0.

21 January 2018

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Crossref

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Herpes simplex virus recovery in neural tissues after ocular HSV shedding induced by epinephrine iontophoresis to the rabbit cornea

21 January 2018

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Iontophoresis of epinephrine isomers to rabbit eyes induced HSV-1 ocular shedding

30 October 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21079815

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

RNA complementary to a herpesvirus alpha gene mRNA is prominent in latently infected neurons

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21079815

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Adrenergic induction of HSV-1 ocular shedding in rabbits

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21079815

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1371/JOURNAL.PONE.0015416

1 reference

29 July 2017

0 references

1 reference

29 July 2017

1 reference