Role of ICAM-1 (CD54) in the development of murine cerebral malaria (Q33331078)

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title

Role of ICAM-1 (CD54) in the development of murine cerebral malaria (English)

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25 July 2017

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R Lucas

7

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author name string

N Favre

1

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C Da Laperousaz

2

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B Ryffel

3

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N A Weiss

4

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B A Imhof

5

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W Rudin

6

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P F Piguet

8

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publication date

1 October 1999

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25 July 2017

published in

Microbes and Infection

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volume

1

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issue

12

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page(s)

961-968

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Tumor necrosis factor (cachectin) as an essential mediator in murine cerebral malaria

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cites work

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Monoclonal antibody against interferon gamma can prevent experimental cerebral malaria and its associated overproduction of tumor necrosis factor

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Interferon-gamma is essential for the development of cerebral malaria

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Respective role of TNF receptors in the development of experimental cerebral malaria.

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Late administration of monoclonal antibody to leukocyte function-antigen 1 abrogates incipient murine cerebral malaria

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Late treatment with anti-LFA-1 (CD11a) antibody prevents cerebral malaria in a mouse model

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Cerebral malaria: mediators, mechanical obstruction or more?

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Leukocytosis and resistance to septic shock in intercellular adhesion molecule 1-deficient mice.

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Crucial role of tumor necrosis factor (TNF) receptor 2 and membrane‐bound TNF in experimental cerebral malaria

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Tumor necrosis factor/cachectin plays a key role in bleomycin-induced pneumopathy and fibrosis

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Lung myeloperoxidase as a measure of pulmonary leukostasis in rabbits

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The development of murine cerebral malaria does not require nitric oxide production

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In vivo sequestration of Plasmodium falciparum-infected human erythrocytes: a severe combined immunodeficiency mouse model for cerebral malaria

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Inflammatory and immune responses are impaired in mice deficient in intercellular adhesion molecule 1.

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LFA-1-deficient mice show normal CTL responses to virus but fail to reject immunogenic tumor

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Urokinase receptor (uPAR, CD87) is a platelet receptor important for kinetics and TNF-induced endothelial adhesion in mice

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Neutrophil emigration in the skin, lungs, and peritoneum: different requirements for CD11/CD18 revealed by CD18-deficient mice

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Stimulation of thrombocytopoiesis decreases platelet β2 but not β1 or β3 integrins

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Role of platelet adhesion in homeostasis and immunopathology

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Recombinant tumor necrosis factor increases pulmonary vascular permeability independent of neutrophils

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Identifiers

DOI

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25 July 2017

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