Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme (Q29547201)

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6

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author name string

Muramatsu M

1

1 reference

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23 April 2017

Kinoshita K

2

1 reference

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Făgărășan S

3

1 reference

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23 April 2017

Yamada S

4

1 reference

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23 April 2017

Shinkai Y

5

1 reference

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language of work or name

1 reference

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publication date

1 September 2000

1 reference

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1 reference

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volume

102

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issue

5

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page(s)

553-63

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Antibody engineering for the analysis of affinity maturation of an anti-hapten response.

23 April 2017

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Unique and unprecedented recombination mechanisms in class switching

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Target specificity of immunoglobulin class switch recombination is not determined by nucleotide sequences of S regions

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Molecular mechanism of immunoglobulin class switch recombination

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Expression of I mu-C gamma hybrid germline transcripts subsequent to immunoglobulin heavy chain class switching

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Within germinal centers, isotype switching of immunoglobulin genes occurs after the onset of somatic mutation

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Ku70 is required for late B cell development and immunoglobulin heavy chain class switching

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Switch circular DNA formed in cytokine-treated mouse splenocytes: evidence for intramolecular DNA deletion in immunoglobulin class switching

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Molecular cloning of apobec-1 complementation factor, a novel RNA-binding protein involved in the editing of apolipoprotein B mRNA.

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Specific expression of activation-induced cytidine deaminase (AID), a novel member of the RNA-editing deaminase family in germinal center B cells

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High frequency class switching of an IgM B lymphoma clone CH12F3 to IgA cells.

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V(D)J recombination: on the cutting edge

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Somatic hypermutation of immunoglobulin genes is linked to transcription initiation

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Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2)

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=11007474

PubMed publication ID

2 references

23 April 2017

Consolidated OpenCitations Corpus – April 2017