Absence epilepsy in tottering mutant mice is associated with calcium channel defects (Q28587452)

31 January 2017

title

Absence epilepsy in tottering mutant mice is associated with calcium channel defects (English)

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Calcium channel, voltage-dependent, P/Q type, alpha 1A subunit

31 January 2017

main subject

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GOA release 2020-03-11

calcium

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author name string

C. F. Fletcher

1

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31 January 2017

C. M. Lutz

2

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31 January 2017

T. N. O'Sullivan

3

1 reference

31 January 2017

J. D. Shaughnessy

4

1 reference

31 January 2017

R. Hawkes

5

1 reference

31 January 2017

W. N. Frankel

6

1 reference

31 January 2017

N. G. Copeland

7

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31 January 2017

N. A. Jenkins

8

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31 January 2017

language of work or name

English

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publication date

15 November 1996

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31 January 2017

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31 January 2017

volume

87

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31 January 2017

issue

4

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31 January 2017

page(s)

607–617

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Expression of tyrosine hydroxylase in cerebellar Purkinje neurons of the mutant tottering and leaner mouse

31 January 2017

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Apoptosis in cerebellar granule cells is blocked by high KCl, forskolin, and IGF-1 through distinct mechanisms of action: the involvement of intracellular calcium and RNA synthesis

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Nerve growth factor regulates tyrosine hydroxylase gene transcription through a nucleoprotein complex that contains c-Fos

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Tottering--a neuromusclar mutation in the mouse. And its linkage with oligosyndacylism

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The modular cerebellum

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Purkinje cell loss from alternating sagittal zones in the cerebellum of leaner mutant mice

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Anticonvulsant sensitivity of absence seizures in the tottering mutant mouse

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Cerebellar cell degeneration in the leaner mutant mouse

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Tottering and leaner mutations perturb transient developmental expression of tyrosine hydroxylase in embryologically distinct purkinje cells

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Mutant mouse tottering: selective increase of locus ceruleus axons in a defined single-locus mutation

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017