Menin is required in cranial neural crest for palatogenesis and perinatal viability (Q28513298)

25 January 2017

title

Menin is required in cranial neural crest for palatogenesis and perinatal viability (English)

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Multiple endocrine neoplasia 1

25 January 2017

main subject

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GOA release 2020-03-11

author name string

Kurt A. Engleka

1

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25 January 2017

Meilin Wu

2

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25 January 2017

Maozhen Zhang

3

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25 January 2017

Nicole B. Antonucci

4

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25 January 2017

Jonathan A. Epstein

5

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25 January 2017

language of work or name

English

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publication date

15 November 2007

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25 January 2017

published in

Developmental Biology

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25 January 2017

volume

311

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25 January 2017

issue

2

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25 January 2017

page(s)

524–537

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Gene recombination in postmitotic cells. Targeted expression of Cre recombinase provokes cardiac-restricted, site-specific rearrangement in adult ventricular muscle in vivo.

25 January 2017

cites work

1 reference

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Distribution of menin-occupied regions in chromatin specifies a broad role of menin in transcriptional regulation

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Menin molecular interactions: insights into normal functions and tumorigenesis.

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Genetic ablation of the tumor suppressor menin causes lethality at mid-gestation with defects in multiple organs.

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Heterozygous Men1 mutant mice develop a range of endocrine tumors mimicking multiple endocrine neoplasia type 1

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Conditional inactivation of the MEN1 gene leads to pancreatic and pituitary tumorigenesis but does not affect normal development of these tissues

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Positional cloning of the gene for multiple endocrine neoplasia-type 1

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The tumor suppressor menin regulates hematopoiesis and myeloid transformation by influencing Hox gene expression

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A mouse model of multiple endocrine neoplasia, type 1, develops multiple endocrine tumors

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Modification of gene activity in mouse embryos in utero by a tamoxifen-inducible form of Cre recombinase

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Genetic background has a major effect on the penetrance and severity of craniofacial defects in mice heterozygous for the gene encoding the nucleolar protein Treacle

7 January 2021

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Menin links estrogen receptor activation to histone H3K4 trimethylation

7 January 2021

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Insertion of Cre into the Pax3 locus creates a new allele of Splotch and identifies unexpected Pax3 derivatives

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CDK inhibitors p18(INK4c) and p27(Kip1) mediate two separate pathways to collaboratively suppress pituitary tumorigenesis

7 January 2021

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Functional collaboration between different cyclin-dependent kinase inhibitors suppresses tumor growth with distinct tissue specificity

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Molecular control of secondary palate development

7 January 2021

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Myc-binding-site recognition in the human genome is determined by chromatin context

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Global and Hox-specific roles for the MLL1 methyltransferase

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Defective neural tube closure and anteroposterior patterning in mice lacking the LIM protein LMO4 or its interacting partner Deaf-1

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Rib truncations and fusions in the Sp2H mouse reveal a role for Pax3 in specification of the ventro-lateral and posterior parts of the somite.

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An essential role for Notch in neural crest during cardiovascular development and smooth muscle differentiation.

7 January 2021

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Menin associates with a trithorax family histone methyltransferase complex and with the hoxc8 locus

7 January 2021

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Menin regulates pancreatic islet growth by promoting histone methylation and expression of genes encoding p27Kip1 and p18INK4c

7 January 2021

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Neural crest expression of Cre recombinase directed by the proximal Pax3 promoter in transgenic mice

7 January 2021

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Improved reporter strain for monitoring Cre recombinase-mediated DNA excisions in mice

7 January 2021

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The paired-like homeo box gene MHox is required for early events of skeletogenesis in multiple lineages

7 January 2021

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Molecular genetics of multiple endocrine neoplasia types 1 and 2.

7 January 2021

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Menin and MLL cooperatively regulate expression of cyclin-dependent kinase inhibitors

7 January 2021

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Menin suppresses osteoblast differentiation by antagonizing the AP-1 factor, JunD

7 January 2021

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Germ-line mutations in p27Kip1 cause a multiple endocrine neoplasia syndrome in rats and humans

7 January 2021

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Null mutation of Dlx-2 results in abnormal morphogenesis of proximal first and second branchial arch derivatives and abnormal differentiation in the forebrain

7 January 2021

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Active genes are tri-methylated at K4 of histone H3

7 January 2021

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Msx1 deficient mice exhibit cleft palate and abnormalities of craniofacial and tooth development

7 January 2021

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Homozygous loss of menin is well tolerated in liver, a tissue not affected in MEN1.

7 January 2021

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Genome-wide analysis of menin binding provides insights into MEN1 tumorigenesis

7 January 2021

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Histone H3 lysine 4 methylation patterns in higher eukaryotic genes

7 January 2021

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Functional interaction between tumor suppressor menin and activator of S-phase kinase

7 January 2021

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Menin induces apoptosis in murine embryonic fibroblasts

7 January 2021

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Craniofacial abnormalities resulting from targeted disruption of the murine Sim2 gene

7 January 2021

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Gene targeting reveals a widespread role for the high-mobility-group transcription factor Sox11 in tissue remodeling

7 January 2021

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Generalized lacZ expression with the ROSA26 Cre reporter strain

7 January 2021

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Inactivation of menin, the product of the multiple endocrine neoplasia type 1 gene, inhibits the commitment of multipotential mesenchymal stem cells into the osteoblast lineage

7 January 2021

1 reference

https://api.crossref.org/works/10.1016/J.YDBIO.2007.08.057

Menin is required for bone morphogenetic protein 2- and transforming growth factor beta-regulated osteoblastic differentiation through interaction with Smads and Runx2

7 January 2021

1 reference

https://api.crossref.org/works/10.1016/J.YDBIO.2007.08.057

7 January 2021

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Leukemia proto-oncoprotein MLL forms a SET1-like histone methyltransferase complex with menin to regulate Hox gene expression

7 January 2021

1 reference

https://api.crossref.org/works/10.1016/J.YDBIO.2007.08.057

The menin tumor suppressor protein is an essential oncogenic cofactor for MLL-associated leukemogenesis

7 January 2021

1 reference

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Altered cell differentiation and proliferation in mice lacking p57KIP2 indicates a role in Beckwith–Wiedemann syndrome

7 January 2021

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10.1016/J.YDBIO.2007.08.057

7 January 2021

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