Chromium(VI) inhibits the transcriptional activity of nuclear factor-kappaB by decreasing the interaction of p65 with cAMP-responsive element-binding protein-binding protein (Q28377525)

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English	Chromium(VI) inhibits the transcriptional activity of nuclear factor-kappaB by decreasing the interaction of p65 with cAMP-responsive element-binding protein-binding protein	scientific article

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Chromium(VI) inhibits the transcriptional activity of nuclear factor-kappaB by decreasing the interaction of p65 with cAMP-responsive element-binding protein-binding protein (English)

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author name string

J A Shumilla

1

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R J Broderick

2

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Y Wang

3

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A Barchowsky

4

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language of work or name

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publication date

17 December 1999

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Journal of Biological Chemistry

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274

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51

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36207-12

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Differential effects of chromium(VI) on constitutive and inducible gene expression in chick embryo liver in vivo and correlation with chromium(VI)-induced DNA damage

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Inhibition of protein synthesis by chromium(VI) differentially affects expression of urokinase and its receptor in human type II pneumocytes

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The transcriptional activity of NF-kappaB is regulated by the IkappaB-associated PKAc subunit through a cyclic AMP-independent mechanism

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Phosphorylation of NF-kappa B p65 by PKA stimulates transcriptional activity by promoting a novel bivalent interaction with the coactivator CBP/p300

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CREB-binding protein/p300 are transcriptional coactivators of p65

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https://api.crossref.org/works/10.1074/JBC.274.51.36207

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Nuclear protein CBP is a coactivator for the transcription factor CREB

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Phosphorylated CREB binds specifically to the nuclear protein CBP

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Activation of cAMP and mitogen responsive genes relies on a common nuclear factor

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A CBP integrator complex mediates transcriptional activation and AP-1 inhibition by nuclear receptors

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Independent modes of transcriptional activation by the p50 and p65 subunits of NF-kappa B

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Arsenic induces oxidant stress and NF-KB activation in cultured aortic endothelial cells

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Inhibition of NF-κB Binding to DNA by Chromium, Cadmium, Mercury, Zinc, and Arsenitein Vitro:Evidence of a Thiol Mechanism

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Effects of Cr(VI) on the expression of the oxidative stress genes in human lung cells

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Mechanism of chromium(VI) carcinogenesis. Reactive intermediates and effect on gene expression

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Transcription factors NF-IL6 and NF-kappa B synergistically activate transcription of the inflammatory cytokines, interleukin 6 and interleukin 8

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One-electron reduction of chromium(VI) by alpha-lipoic acid and related hydroxyl radical generation, dG hydroxylation and nuclear transcription factor-kB activation

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Chromium(VI) treatment of normal human lung cells results in guanine-specific DNA polymerase arrest, DNA-DNA cross-links and S-phase blockade of cell cycle

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Metal-induced modulation of nitric oxide production in vitro by murine macrophages: lead, nickel, and cobalt utilize different mechanisms

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Alterations in the prooxidant and antioxidant status of human leukemic T-lymphocyte MOLT4 cells treated with potassium chromate

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A p300/CBP-associated factor that competes with the adenoviral oncoprotein E1A

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Characterization of DNA-protein complexes induced in intact cells by the carcinogen chromate.

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Immunological detection of DNA-protein complexes induced by chromate.

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Steroid receptor coactivator-1 coactivates activating protein-1-mediated transactivations through interaction with the c-Jun and c-Fos subunits

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Nuclear hormone receptor antagonism with AP-1 by inhibition of the JNK pathway

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Stimulation of urokinase expression by TNF-alpha requires the activation of binding sites for the AP-1 and PEA3 transcription factors

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21 January 2018

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10.1074/JBC.274.51.36207

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