Faciobrachial dystonic seizures precede Lgi1 antibody limbic encephalitis (Q28307624)

9

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Sarosh R Irani

object named as

Sarosh R Irani

1

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author name string

Andrew W Michell

2

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Bethan Lang

3

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Philippa Pettingill

4

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Patrick Waters

5

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Michael R Johnson

6

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Jonathan M Schott

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Richard J E Armstrong

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Andrew Bleasel

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Ernest R Somerville

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Shelagh M J Smith

Potassium channel antibody-associated encephalopathy: a potentially immunotherapy-responsive form of limbic encephalitis

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language of work or name

English

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publication date

May 2011

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published in

Annals of Neurology

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volume

69

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issue

5

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page(s)

892-900

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cites work

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Potentially reversible autoimmune limbic encephalitis with neuronal potassium channel antibody

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Antibodies to Kv1 potassium channel-complex proteins leucine-rich, glioma inactivated 1 protein and contactin-associated protein-2 in limbic encephalitis, Morvan's syndrome and acquired neuromyotonia

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Investigation of LGI1 as the antigen in limbic encephalitis previously attributed to potassium channels: a case series

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Antibodies to glutamic acid decarboxylase define a form of limbic encephalitis

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FDG-PET and MRI in potassium channel antibody-associated non-paraneoplastic limbic encephalitis: correlation with clinical course and neuropsychology

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Clinical spectrum of voltage-gated potassium channel autoimmunity

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Arrested maturation of excitatory synapses in autosomal dominant lateral temporal lobe epilepsy.

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Mutations in the LGI1/Epitempin gene on 10q24 cause autosomal dominant lateral temporal epilepsy

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LGI1 is mutated in familial temporal lobe epilepsy characterized by aphasic seizures

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Mutations in LGI1 cause autosomal-dominant partial epilepsy with auditory features

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