Atrial arrhythmias in inherited arrhythmogenic disorders (Q28076679)

language of work or name

1 reference

October 2016

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published in

Journal of arrhythmia

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volume

32

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issue

5

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page(s)

366-372

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Mutations in SCN10A are responsible for a large fraction of cases of Brugada syndrome

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Loss-of-function mutations in the cardiac calcium channel underlie a new clinical entity characterized by ST-segment elevation, short QT intervals, and sudden cardiac death

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Identification of a novel loss-of-function calcium channel gene mutation in short QT syndrome (SQTS6)

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Common and rare variants in SCN10A modulate the risk of atrial fibrillation

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p.Y1449C SCN5A mutation associated with overlap disorder comprising conduction disease, Brugada syndrome, and atrial flutter

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Early repolarization as a predictor of arrhythmic and nonarrhythmic cardiac events in middle-aged subjects

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Calsequestrin 2 deletion causes sinoatrial node dysfunction and atrial arrhythmias associated with altered sarcoplasmic reticulum calcium cycling and degenerative fibrosis within the mouse atrial pacemaker complex1.

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Distribution and function of sodium channel subtypes in human atrial myocardium

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KCNJ2 mutation in short QT syndrome 3 results in atrial fibrillation and ventricular proarrhythmia

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Long-term follow-up of a pediatric cohort with short QT syndrome

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Prevalence, clinical characteristics and management of atrial fibrillation in patients with Brugada syndrome

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A KCNJ8 mutation associated with early repolarization and atrial fibrillation

29 September 2017

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Genetic, molecular and cellular mechanisms underlying the J wave syndromes

29 September 2017

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29 September 2017

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Molecular genetic and functional association of Brugada and early repolarization syndromes with S422L missense mutation in KCNJ8

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Phenotypic Manifestations of Mutations in Genes Encoding Subunits of Cardiac Potassium Channels

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Mutations in the cardiac L-type calcium channel associated with inherited J-wave syndromes and sudden cardiac death.

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An international compendium of mutations in the SCN5A-encoded cardiac sodium channel in patients referred for Brugada syndrome genetic testing

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A mutation in the beta 3 subunit of the cardiac sodium channel associated with Brugada ECG phenotype

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Prevalence of supraventricular tachyarrhythmias in a cohort of 115 patients with Brugada syndrome

29 September 2017

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Prevalence of early-onset atrial fibrillation in congenital long QT syndrome

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Novel pore mutation in SCN5A manifests as a spectrum of phenotypes ranging from atrial flutter, conduction disease, and Brugada syndrome to sudden cardiac death

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Sick Sinus Syndrome, Progressive Cardiac Conduction Disease, Atrial Flutter and Ventricular Tachycardia Caused by a Novel SCN5A Mutation

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Right praecordial early repolarization pattern occurring with the Wolff-Parkinson-White pattern

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Atrial fibrillation and atrial vulnerability in patients with Brugada syndrome

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Brugada syndrome caused by a large deletion in SCN5A only detected by multiplex ligation-dependent probe amplification

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Early Repolarization Pattern Occurring with the Wolff-Parkinson-White Syndrome

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Increased Intraatrial Conduction Abnormality Assessed by P‐Wave Signal‐Averaged Electrocardiogram in Patients with Brugada Syndrome

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Efficacy of pulmonary vein isolation in paroxysmal atrial fibrillation patients with a Brugada electrocardiogram

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Preexcitation unmasks J waves: 2 cases

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Atrial fibrillation in patients with Brugada syndrome relationships of gene mutation, electrophysiology, and clinical backgrounds

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Identifiers

DOI

0 references

1 reference

PubMed publication ID

27761160

1 reference