PKR and RNase L Contribute to Protection against Lethal West Nile Virus Infection by Controlling Early Viral Spread in the Periphery and Replication in Neurons (Q27473291)

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scientific article (publication date: July 2006)

Language	Label	Description	Also known as
English	PKR and RNase L Contribute to Protection against Lethal West Nile Virus Infection by Controlling Early Viral Spread in the Periphery and Replication in Neurons	scientific article (publication date: July 2006)

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scholarly article

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PKR and RNase L Contribute to Protection against Lethal West Nile Virus Infection by Controlling Early Viral Spread in the Periphery and Replication in Neurons (English)

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West Nile virus

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Michael S. Diamond

9

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Winfried Barchet

object named as

Winfried Barchet

5

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object named as

Michael Gale

8

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object named as

Brian C Keller

3

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Robert H. Silverman

7

object named as

Robert H Silverman

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Melanie A. Samuel

1

object named as

Melanie A Samuel

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author name string

Kevin Whitby

2

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Anantha Marri

4

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Bryan R G Williams

6

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publication date

July 2006

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Journal of Virology

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80

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14

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7009-19

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RNase L plays a role in the antiviral response to West Nile virus

1 reference

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West Nile virus evades activation of interferon regulatory factor 3 through RIG-I-dependent and -independent pathways without antagonizing host defense signaling

1 reference

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A Single Amino Acid Substitution in the West Nile Virus Nonstructural Protein NS2A Disables Its Ability To Inhibit Alpha/Beta Interferon Induction and Attenuates Virus Virulence in Mice

1 reference

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CCR5 deficiency increases risk of symptomatic West Nile virus infection

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Chemokine receptor CCR5 promotes leukocyte trafficking to the brain and survival in West Nile virus infection

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Fatal fulminant pan-meningo-polioencephalitis due to West Nile virus

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Signal integration via PKR.

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