p37δ is a new isoform of PI3K p110δ that increases cell proliferation and is overexpressed in tumors (Q24294459)

GOA release 2020-03-11

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S Fransson

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A Uv

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H Eriksson

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M K Andersson

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Y Wettergren

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M Bergo

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K Ejeskär

The p85 regulatory subunit of phosphoinositide 3-kinase down-regulates IRS-1 signaling via the formation of a sequestration complex

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language of work or name

English

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publication date

5 July 2012

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published in

Oncogene

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volume

31

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issue

27

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page(s)

3277-86

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cites work

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Key role of the p110delta isoform of PI3K in B-cell antigen and IL-4 receptor signaling: comparative analysis of genetic and pharmacologic interference with p110delta function in B cells

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Direct positive regulation of PTEN by the p85 subunit of phosphatidylinositol 3-kinase

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Alteration of gene expression in normal-appearing colon mucosa of APC(min) mice and human cancer patients

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Phosphoinositide 3-kinase p110beta activity: key role in metabolism and mammary gland cancer but not development

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Activity of any class IA PI3K isoform can sustain cell proliferation and survival.

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Oncogenic transformation induced by the p110beta, -gamma, and -delta isoforms of class I phosphoinositide 3-kinase

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Regulation of p110delta PI 3-kinase gene expression

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The PI3K p110delta controls T-cell development, differentiation and regulation

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Proteomic analysis reveals field-wide changes in protein expression in the morphologically normal mucosa of patients with colorectal neoplasia

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Cloning of PI3 kinase-associated p85 utilizing a novel method for expression/cloning of target proteins for receptor tyrosine kinases

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Essential role for the p110 isoform in phosphoinositide 3-kinase activation and cell proliferation in acute myeloid leukemia

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The phosphoinositide 3-kinase regulatory subunit p85alpha can exert tumor suppressor properties through negative regulation of growth factor signaling

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Signalling by PI3K isoforms: insights from gene-targeted mice.

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PI 3-kinase and cancer: changing accents

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p16INK4a gene promoter hypermethylation in mucosa as a prognostic factor for patients with colorectal cancer.

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Regulation of the p85/p110 phosphatidylinositol 3'-kinase: stabilization and inhibition of the p110alpha catalytic subunit by the p85 regulatory subunit

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Class I PI3K in oncogenic cellular transformation.

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Neuroblastoma tumors with favorable and unfavorable outcomes: Significant differences in mRNA expression of genes mapped at 1p36.2.

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Essential roles of PI(3)K-p110beta in cell growth, metabolism and tumorigenesis

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Phosphatidylinositol (3,4,5)P3 interacts with SH2 domains and modulates PI 3-kinase association with tyrosine-phosphorylated proteins

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