USP47 is a deubiquitylating enzyme that regulates base excision repair by controlling steady-state levels of DNA polymerase β (Q24294446)

Ubiquitin specific peptidase 47

GOA release 2020-03-11

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monoubiquitinated protein deubiquitination

GOA release 2020-03-11

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GOA release 2020-03-11

Mariola J Edelmann

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Benedikt M. Kessler

object named as

Benedikt M Kessler

5

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Jason L Parsons

object named as

Jason L Parsons

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author name string

Irina I Dianova

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Svetlana V Khoronenkova

3

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Grigory L Dianov

Cloning and enzymatic analysis of 22 novel human ubiquitin-specific proteases

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language of work or name

English

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publication date

4 March 2011

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published in

Molecular Cell

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volume

41

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issue

5

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page(s)

609-15

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cites work

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The ARF tumour suppressor

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ARF-BP1/Mule is a critical mediator of the ARF tumor suppressor

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Co-ordination of DNA single strand break repair

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Poly ADP-ribose polymerase-1: an international molecule of mystery

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Poly(ADP-ribose) polymerase-1 modulates DNA repair capacity and prevents formation of DNA double strand breaks

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CHIP-mediated degradation and DNA damage-dependent stabilization regulate base excision repair proteins.

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A semi-automated large-scale process for the production of recombinant tagged proteins in the Baculovirus expression system

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Instability and decay of the primary structure of DNA

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Requirement of mammalian DNA polymerase-beta in base-excision repair.

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XRCC1 and DNA polymerase beta in cellular protection against cytotoxic DNA single-strand breaks

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Ubiquitin ligase ARF-BP1/Mule modulates base excision repair

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Regulation of monoubiquitinated PCNA by DUB autocleavage

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The ubiquitin-specific protease USP47 is a novel beta-TRCP interactor regulating cell survival

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Protein partners of deubiquitinating enzymes

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Two Pathways for Base Excision Repair in Mammalian Cells

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Role of DNA polymerase beta in the excision step of long patch mammalian base excision repair

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DNA polymerase beta is the major dRP lyase involved in repair of oxidative base lesions in DNA by mammalian cell extracts

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Human DNA polymerase beta initiates DNA synthesis during long-patch repair of reduced AP sites in DNA

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Overexpression of DNA polymerase beta results in an increased rate of frameshift mutations during base excision repair

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Overexpression of DNA polymerase β: a genomic instability enhancer process

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DNA damage disrupts the p14ARF-B23(nucleophosmin) interaction and triggers a transient subnuclear redistribution of p14ARF.

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