Possible cancer-causing capacity of COVID-19: Is SARS-CoV-2 an oncogenic agent? (Q131604203)

https://api.crossref.org/v1/works/https://doi.org/10.1016/j.biochi.2023.05.014

31 December 2024

title

Possible cancer-causing capacity of COVID-19: Is SARS-CoV-2 an oncogenic agent? (English)

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https://api.crossref.org/v1/works/https://doi.org/10.1016/j.biochi.2023.05.014

31 December 2024

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SARS-CoV-2

31 December 2024

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COVID-19

31 December 2024

Kasra Jahankhani

1

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https://api.crossref.org/v1/works/https://doi.org/10.1016/j.biochi.2023.05.014

31 December 2024

Fatemeh Ahangari

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2

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https://api.crossref.org/v1/works/https://doi.org/10.1016/j.biochi.2023.05.014

31 December 2024

Ian M. Adcock

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3

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https://api.crossref.org/v1/works/https://doi.org/10.1016/j.biochi.2023.05.014

31 December 2024

Esmaeil Mortaz

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4

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31 December 2024

publication date

October 2023

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31 December 2024

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31 December 2024

volume

213

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31 December 2024

page(s)

130-138

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The risk of pancreatic adenocarcinoma following SARS-CoV family infection

31 December 2024

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Targeting Stat3 blocks both HIF-1 and VEGF expression induced by multiple oncogenic growth signaling pathways

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Cancer progression in COVID-19: integrating the roles of renin angiotensin aldosterone system, angiopoietin-2, heat shock protein-27 and epithelial mesenchymal transition

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IL-17 enhances the net angiogenic activity and in vivo growth of human non-small cell lung cancer in SCID mice through promoting CXCR-2-dependent angiogenesis

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Angiotensin receptors: a new role in cancer?

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Mechanism of Ang II involvement in activation of NF-κB through phosphorylation of p65 during aging

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STAT3 orchestrates contradictory signals in cytokine-induced G1 to S cell-cycle transition

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Biological behavior of oral squamous cell carcinoma in the background of novel corona virus infection

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Epigenetic down-regulated DDX10 promotes cell proliferation through Akt/NF-κB pathway in ovarian cancer

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EMMPRIN/BASIGIN as a biological modulator of oral cancer and COVID-19 interaction: Novel propositions

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Interleukin-6 promotes cervical tumor growth by VEGF-dependent angiogenesis via a STAT3 pathway

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Angiotensin-(1-7): beyond the cardio-renal actions.

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10.1016/J.BIOCHI.2023.05.014

1 reference

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