CCL11
Внешний вид
CCL11 (ингл. ) — аксымы, шул ук исемдәге ген тарафыннан кодлана торган югары молекуляр органик матдә.[21][22]
Искәрмәләр
[үзгәртү | вики-текстны үзгәртү]- ↑ 1,0 1,1 UniProt
- ↑ 2,00 2,01 2,02 2,03 2,04 2,05 2,06 2,07 2,08 2,09 2,10 2,11 2,12 2,13 2,14 2,15 2,16 2,17 2,18 2,19 2,20 2,21 2,22 GOA
- ↑ Hundelshausen P. V., Grommes J., Weber C. et al. Chemokine interactome mapping enables tailored intervention in acute and chronic inflammation // Sci. Transl. Med. — AAAS, 2017. — ISSN 1946-6234; 1946-6242 — doi:10.1126/SCITRANSLMED.AAH6650 — PMID:28381538
- ↑ Smet F. D., Bock K. D., Georgiadou M. et al. Inhibition of tumor angiogenesis and growth by a small-molecule multi-FGF receptor blocker with allosteric properties // Cancer Cell — Cell Press, Elsevier BV, 2013. — ISSN 1535-6108; 1878-3686 — doi:10.1016/J.CCR.2013.02.019 — PMID:23597562
- ↑ Gorrell M. D. Neuropeptide Y, B-type natriuretic peptide, substance P and peptide YY are novel substrates of fibroblast activation protein-α // FEBS J. — Wiley-Blackwell, 2011. — ISSN 1742-464X; 0014-2956; 1742-4658; 1432-1033 — doi:10.1111/J.1742-4658.2011.08051.X — PMID:21314817
- ↑ 6,0 6,1 6,2 6,3 6,4 GOA
- ↑ 7,0 7,1 Pease J. E. Differential regulation of eosinophil chemokine signaling via CCR3 and non-CCR3 pathways // J. Immunol. — Baltimore: 1999. — ISSN 0022-1767; 1550-6606 — PMID:10072545
- ↑ 8,0 8,1 J Ye, Mayer K. L., Mayer M. R. et al. NMR solution structure and backbone dynamics of the CC chemokine eotaxin-3 // Biochemistry / A. Schepartz — ACS, 2001. — ISSN 0006-2960; 1520-4995; 1943-295X — doi:10.1021/BI010252S — PMID:11425309
- ↑ Crump M. P. Solution structure of eotaxin, a chemokine that selectively recruits eosinophils in allergic inflammation // J. Biol. Chem. / L. M. Gierasch — Baltimore [etc.]: American Society for Biochemistry and Molecular Biology, 1998. — ISSN 0021-9258; 1083-351X; 1067-8816 — doi:10.1074/JBC.273.35.22471 — PMID:9712872
- ↑ 10,0 10,1 10,2 10,3 Pease J. E. Differential regulation of eosinophil chemokine signaling via CCR3 and non-CCR3 pathways // J. Immunol. — Baltimore: 1999. — ISSN 0022-1767; 1550-6606 — PMID:10072545
- ↑ 11,00 11,01 11,02 11,03 11,04 11,05 11,06 11,07 11,08 11,09 11,10 11,11 11,12 11,13 Livstone M. S., Thomas P. D., Lewis S. E. et al. Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium // Brief. Bioinform. — OUP, 2011. — ISSN 1467-5463; 1477-4054 — doi:10.1093/BIB/BBR042 — PMID:21873635
- ↑ 12,0 12,1 12,2 12,3 Nozaki M., Smith J., Humbles A. A. et al. CCR3 is a target for age-related macular degeneration diagnosis and therapy // Nature / M. Skipper — NPG, Springer Science Business Media, 2009. — ISSN 1476-4687; 0028-0836 — doi:10.1038/NATURE08151 — PMID:19525930
- ↑ Fauci A. S., M Dybul, AS F. et al. Dendritic cells express multiple chemokine receptors used as coreceptors for HIV entry // J. Immunol. — Baltimore: 1998. — ISSN 0022-1767; 1550-6606 — PMID:9558100
- ↑ RS K., KC W., Alvarez-Hernandez X et al. Chemokine receptor expression and signaling in macaque and human fetal neurons and astrocytes: implications for the neuropathogenesis of AIDS // J. Immunol. — Baltimore: 1999. — ISSN 0022-1767; 1550-6606 — PMID:10415069
- ↑ 15,0 15,1 15,2 Kampen G. T., S Stafford, T Adachi et al. Eotaxin induces degranulation and chemotaxis of eosinophils through the activation of ERK2 and p38 mitogen-activated protein kinases // Blood — American Society of Hematology, Elsevier BV, 2000. — ISSN 0006-4971; 1528-0020 — PMID:10706854
- ↑ Nozaki M., Smith J., Humbles A. A. et al. CCR3 is a target for age-related macular degeneration diagnosis and therapy // Nature / M. Skipper — NPG, Springer Science Business Media, 2009. — ISSN 1476-4687; 0028-0836 — doi:10.1038/NATURE08151 — PMID:19525930
- ↑ T Jinquan, S Quan, G Feili et al. Eotaxin activates T cells to chemotaxis and adhesion only if induced to express CCR3 by IL-2 together with IL-4 // J. Immunol. — Baltimore: 1999. — ISSN 0022-1767; 1550-6606 — PMID:10201960
- ↑ 18,0 18,1 Huber M. A., N Kraut, T Addicks et al. Cell-type-dependent induction of eotaxin and CCR3 by ionizing radiation // Biochem. Biophys. Res. Commun. — Academic Press, Elsevier BV, 2000. — ISSN 0006-291X; 1090-2104 — doi:10.1006/BBRC.2000.2287 — PMID:10708591
- ↑ Huber M. A., N Kraut, T Addicks et al. Cell-type-dependent induction of eotaxin and CCR3 by ionizing radiation // Biochem. Biophys. Res. Commun. — Academic Press, Elsevier BV, 2000. — ISSN 0006-291X; 1090-2104 — doi:10.1006/BBRC.2000.2287 — PMID:10708591
- ↑ Yang D., Chen Q., Hoover D. M. et al. Many chemokines including CCL20/MIP-3alpha display antimicrobial activity. // J. Leukoc. Biol. / L. Montaner — Wiley-Blackwell, 2003. — ISSN 0741-5400; 1938-3673 — doi:10.1189/JLB.0103024 — PMID:12949249
- ↑ HUGO Gene Nomenclature Commitee, HGNC:29223 (ингл.). әлеге чыганактан 2015-10-25 архивланды. 18 сентябрь, 2017 тикшерелгән.
- ↑ UniProt, Q9ULJ7 (ингл.). 18 сентябрь, 2017 тикшерелгән.
Чыганаклар
[үзгәртү | вики-текстны үзгәртү]- Степанов В.М. (2005). Молекулярная биология. Структура и функция белков. Москва: Наука. ISBN 5-211-04971-3.(рус.)
- Bruce Alberts, Alexander Johnson, Julian Lewis, Martin Raff, Keith Roberts, Peter Walter (2002). Molecular Biology of the Cell (вид. 4th). Garland. ISBN 0815332181.(ингл.)
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