(Q55473556)

12 July 2018

title

Inhibition of TRAIL-induced apoptosis by Bcl-2 overexpression. (English)

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12 July 2018

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1

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12 July 2018

Klaus-Michael Debatin

series ordinal

3

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12 July 2018

author name string

Eric Meyer

series ordinal

2

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12 July 2018

language of work or name

English

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publication date

1 April 2002

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12 July 2018

published in

Oncogene

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12 July 2018

volume

21

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12 July 2018

issue

15

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12 July 2018

page(s)

2283-2294

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12 July 2018

Cytotoxic Drugs, Programmed Cell Death, and the Immune System: Defining New Roles in an Old Play

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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Bcl-2 expression in higher-grade human glioma: a clinical and experimental study

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Activation of the CD95 (APO-1/Fas) pathway in drug- and gamma-irradiation-induced apoptosis of brain tumor cells

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The biochemistry of apoptosis

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Tumoricidal activity of a novel anti-human DR5 monoclonal antibody without hepatocyte cytotoxicity.

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The proto-oncogene Bcl-2 and its role in regulating apoptosis

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Mitochondrial control of cell death

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FLICE is activated by association with the CD95 death-inducing signaling complex (DISC).

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The potential of TRAIL for cancer chemotherapy

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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APO2 ligand: a novel lethal weapon against malignant glioma?

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Bcl-2 oncoprotein protects the human prostatic carcinoma cell line PC3 from TRAIL-mediated apoptosis.

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FLICE is predominantly expressed as two functionally active isoforms, caspase-8/a and caspase-8/b.

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Executioner caspase-3, -6, and -7 perform distinct, non-redundant roles during the demolition phase of apoptosis.

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FADD/MORT1 and caspase-8 are recruited to TRAIL receptors 1 and 2 and are essential for apoptosis mediated by TRAIL receptor 2

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The CD95 (APO-1/Fas) and the TRAIL (APO-2L) apoptosis systems

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Caspase-3 is the primary activator of apoptotic DNA fragmentation via DNA fragmentation factor-45/inhibitor of caspase-activated DNase inactivation.

21 January 2018

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The role of chemotherapy in the treatment of children with neuroblastoma stage IV: the GPO (German Pediatric Oncology Society) experience

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7 January 2021

Apoptosis-induced by TRAIL AND TNF-alpha in human multiple myeloma cells is not blocked by BCL-2

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7 January 2021

Bcl-XL protects pancreatic adenocarcinoma cells against CD95- and TRAIL-receptor-mediated apoptosis

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7 January 2021

Cellular death in neuroblastoma: in situ correlation of apoptosis and bcl‐2 expression

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7 January 2021

XIAP, the guardian angel

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7 January 2021

Failure of Bcl-2 to block cytochrome c redistribution during TRAIL-induced apoptosis

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7 January 2021

Differential hepatocyte toxicity of recombinant Apo2L/TRAIL versions

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7 January 2021

Mechanisms of CD95 (APO-1/Fas)-mediated apoptosis

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7 January 2021

Overexpression of BCL2 blocks TNF-related apoptosis-inducing ligand (TRAIL)-induced apoptosis in human lung cancer cells

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1205258

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12 July 2018

Dimensions Publication ID

1027213673

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12 July 2018