(Q48902327)

16 February 2018

title

Disturbances of calcium homeostasis within the endoplasmic reticulum may contribute to the development of ischemic-cell damage (English)

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16 February 2018

main subject

endoplasmic reticulum

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author name string

Paschen W

series ordinal

1

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16 February 2018

publication date

1 October 1996

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16 February 2018

published in

Medical Hypotheses

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16 February 2018

volume

47

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16 February 2018

issue

4

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16 February 2018

page(s)

283-288

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16 February 2018

Cell damage in the brain: a speculative synthesis

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Historical overview. Calcium, ischemia, and death of brain cells

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Calcium fluxes, calcium antagonists, and calcium-related pathology in brain ischemia, hypoglycemia, and spreading depression: a unifying hypothesis

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Changes in Extracellular Calcium Activity in Cerebral Ischaemia

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Cation activities in reversible ischemia of the cat brain

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Origin of intracellular Ca2 elevation induced by in vitro ischemia-like condition in hippocampal slices

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Changes in brain energy metabolism and protein synthesis following transient bilateral ischemia in the gerbil

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Barbiturate promotes post-ischemic reaggregation of polyribosomes in gerbil hippocampus

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Stress-induced inhibition of protein synthesis initiation: modulation of initiation factor 2 and guanine nucleotide exchange factor activities following transient cerebral ischemia in the rat.

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Calcium store depletion in dimethyl BAPTA-loaded human platelets increases protein tyrosine phosphorylation in the absence of a rise in cytosolic calcium

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Thapsigargin, a Ca(2 )-ATPase inhibitor, depletes the intracellular Ca2 pool and induces apoptosis in human hepatoma cells

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Internucleosomal DNA cleavage involved in ischemia-induced neuronal death

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The microsomal calcium-ATPase inhibitor thapsigargin is a neurotoxin in perinatal rodent brain

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Identifiers

DOI

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16 February 2018

1 reference

16 February 2018