(Q40722488)

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The leukemia-associated transcription repressor AML1/MDS1/EVI1 requires CtBP to induce abnormal growth and differentiation of murine hematopoietic cells

scientific article published on May 2002

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Europe PubMed Central

PubMed publication ID

23 September 2017

The leukemia-associated transcription repressor AML1/MDS1/EVI1 requires CtBP to induce abnormal growth and differentiation of murine hematopoietic cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

author name string

Vitalyi Senyuk

1

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Soumen Chakraborty

2

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Fady M Mikhail

3

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Rui Zhao

4

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Yiqing Chi

5

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Giuseppina Nucifora

6

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Europe PubMed Central

PubMed publication ID

23 September 2017

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publication date

1 May 2002

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Europe PubMed Central

PubMed publication ID

23 September 2017

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

21

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Europe PubMed Central

PubMed publication ID

23 September 2017

20

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

3232-3240

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

ETO, a target of t(8;21) in acute leukemia, makes distinct contacts with multiple histone deacetylases and binds mSin3A through its oligomerization domain

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

The leukemia-associated gene TEL encodes a transcription repressor which associates with SMRT and mSin3A.

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Interaction of EVI1 with cAMP-responsive element-binding protein-binding protein (CBP) and p300/CBP-associated factor (P/CAF) results in reversible acetylation of EVI1 and in co-localization in nuclear speckles

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Human AML1/MDS1/EVI1 fusion protein induces an acute myelogenous leukemia (AML) in mice: a model for human AML

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Three proteins define a class of human histone deacetylases related to yeast Hda1p

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Intergenic splicing of MDS1 and EVI1 occurs in normal tissues as well as in myeloid leukemia and produces a new member of the PR domain family

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Diagnostic significance of dysplastic features of peripheral blood polymorphs in myelodysplastic syndromes

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Blimp-1 is the murine homolog of the human transcriptional repressor PRDI-BF1

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

The corepressor CtBP interacts with Evi-1 to repress transforming growth factor beta signaling

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

dCtBP-dependent and -independent repression activities of the Drosophila Knirps protein

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Loss of erythropoietin responsiveness in erythroid progenitors due to expression of the Evi-1 myeloid-transforming gene

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Retrovirus-mediated gene transfer of MLL-ELL transforms primary myeloid progenitors and causes acute myeloid leukemias in mice

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Acquisition of oncogenic potential by RAR chimeras in acute promyelocytic leukemia through formation of homodimers.

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Oligomerization of RAR and AML1 transcription factors as a novel mechanism of oncogenic activation.

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

dCtBP mediates transcriptional repression by Knirps, Krüppel and Snail in the Drosophila embryo

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Consistent intergenic splicing and production of multiple transcripts between AML1 at 21q22 and unrelated genes at 3q26 in (3;21)(q26;q22) translocations

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Molecular cloning and characterization of PEBP2 beta, the heterodimeric partner of a novel Drosophila runt-related DNA binding protein PEBP2 alpha

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

PEBP2/PEA2 represents a family of transcription factors homologous to the products of the Drosophila runt gene and the human AML1 gene

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

AML1, the target of multiple chromosomal translocations in human leukemia, is essential for normal fetal liver hematopoiesis

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Evi-1 transforming and repressor activities are mediated by CtBP co-repressor proteins

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

ZEB represses transcription through interaction with the corepressor CtBP

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Synergistic up-regulation of the myeloid-specific promoter for the macrophage colony-stimulating factor receptor by AML1 and the t(8;21) fusion protein may contribute to leukemogenesis

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Molecular cloning and characterization of a cellular phosphoprotein that interacts with a conserved C-terminal domain of adenovirus E1A involved in negative modulation of oncogenic transformation

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

C-Terminal binding protein is a transcriptional repressor that interacts with a specific class of vertebrate Polycomb proteins

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

The leukemia-associated gene MDS1/EVI1 is a new type of GATA-binding transactivator

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

The carboxy-terminal region of adenovirus E1A activates transcription through targeting of a C-terminal binding protein-histone deacetylase complex

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Cloning and characterization of mCtBP2, a co-repressor that associates with basic Krüppel-like factor and other mammalian transcriptional regulators

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

The CtBP family: enigmatic and enzymatic transcriptional co-repressors

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Disruption of the Cbfa2 gene causes necrosis and hemorrhaging in the central nervous system and blocks definitive hematopoiesis

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

NF-E2p18/mafK is required in DMSO-induced differentiation of Friend erythroleukemia cells by enhancing NF-E2 activity

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

CCAAT enhancer-binding protein (C/EBP) and AML1 (CBF alpha2) synergistically activate the macrophage colony-stimulating factor receptor promoter

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

21 January 2018

Neutrophils with ring-shaped nuclei in myeloproliferative disease

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

7 January 2021

based on heuristic

inferred from DOI database lookup

MDS1/EVI1 enhances TGF-beta1 signaling and strengthens its growth-inhibitory effect but the leukemia-associated fusion protein AML1/MDS1/EVI1, product of the t(3;21), abrogates growth-inhibition in response to TGF-beta1.

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

7 January 2021

based on heuristic

inferred from DOI database lookup

Rearrangement of the AML1/CBFA2 gene in myeloid leukemia with the 3;21 translocation: expression of co-existing multiple chimeric genes with similar functions as transcriptional repressors, but with opposite tumorigenic properties.

1 reference

https://api.crossref.org/works/10.1038/SJ.ONC.1205436

7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1038/SJ.ONC.1205436

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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